Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Anna Shteinfer-Kuzmine

Showing results (31-40 of 37) with videos related to

Pageof 4
Sort By:
You have reached the last page of results.This site can display upto 37 results.
Oncotarget|March 22, 2018
Hypoxic-induced truncation of voltage-dependent anion channel 1 is mediated by both asparagine endopeptidase and calpain 1 activitiesHadas Pahima, Simona Reina, Noa Tadmor, et al.
International Journal of Molecular Sciences|September 9, 2022
Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALSAnna Shteinfer-Kuzmine, Shirel Argueti-Ostrovsky, Marcel F Leyton-Jaimes, et al.
Biomolecules|October 27, 2022
The Multicellular Effects of VDAC1 N-Terminal-Derived PeptideUttpal Anand, Anna Shteinfer-Kuzmine, Gal Sela, et al.
Apoptosis : an International Journal on Programmed Cell Death|October 7, 2024
Elevated serum mtDNA in COVID-19 patients is linked to SARS-CoV-2 envelope protein targeting mitochondrial VDAC1, inducing apoptosis and mtDNA releaseAnna Shteinfer-Kuzmine, Ankit Verma, Rut Bornshten, et al.
Frontiers in Cellular Neuroscience|September 3, 2019
A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALSAnna Shteinfer-Kuzmine, Shirel Argueti, Rajeev Gupta, et al.
Translational Neurodegeneration|December 28, 2022
Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathologyAnkit Verma, Anna Shteinfer-Kuzmine, Nikita Kamenetsky, et al.
Science (New York, N.Y.)|December 21, 2019
VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like diseaseJeonghan Kim, Rajeev Gupta, Luz P Blanco, et al.
Pageof 4

Showing results (31-40 of 37) with videos related to

Sort By:
Pageof 4
You have reached the last page of results.This site can display upto 37 results.
Oncotarget|March 22, 2018
Hypoxic-induced truncation of voltage-dependent anion channel 1 is mediated by both asparagine endopeptidase and calpain 1 activitiesHadas Pahima, Simona Reina, Noa Tadmor, et al.
International Journal of Molecular Sciences|September 9, 2022
Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALSAnna Shteinfer-Kuzmine, Shirel Argueti-Ostrovsky, Marcel F Leyton-Jaimes, et al.
Biomolecules|October 27, 2022
The Multicellular Effects of VDAC1 N-Terminal-Derived PeptideUttpal Anand, Anna Shteinfer-Kuzmine, Gal Sela, et al.
Apoptosis : an International Journal on Programmed Cell Death|October 7, 2024
Elevated serum mtDNA in COVID-19 patients is linked to SARS-CoV-2 envelope protein targeting mitochondrial VDAC1, inducing apoptosis and mtDNA releaseAnna Shteinfer-Kuzmine, Ankit Verma, Rut Bornshten, et al.
Frontiers in Cellular Neuroscience|September 3, 2019
A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALSAnna Shteinfer-Kuzmine, Shirel Argueti, Rajeev Gupta, et al.
Translational Neurodegeneration|December 28, 2022
Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathologyAnkit Verma, Anna Shteinfer-Kuzmine, Nikita Kamenetsky, et al.
Science (New York, N.Y.)|December 21, 2019
VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like diseaseJeonghan Kim, Rajeev Gupta, Luz P Blanco, et al.
Pageof 4