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BMC Cancer|December 4, 2014
Recurrent CYP2C19 deletion allele is associated with triple-negative breast cancerAnna Tervasmäki, Robert Winqvist, Arja Jukkola-Vuorinen, et al.
International Journal of Cancer|September 26, 2022
ATM c.7570G>C is a high-risk allele for breast cancerMinna Kankuri-Tammilehto, Anna Tervasmäki, Minna Kraatari-Tiri, et al.
European Journal of Cancer (Oxford, England : 1990)|December 6, 2020
Exome sequencing identifies a recurrent variant in SERPINA3 associating with hereditary susceptibility to breast cancerSusanna Koivuluoma, Anna Tervasmäki, Saila Kauppila, et al.
Breast Cancer Research and Treatment|February 26, 2025
Population-based study of recurrent DNA damage response gene variants in breast cancer casesAnna Tervasmäki, Timo A Kumpula, Mervi Grip, et al.
Genes, Chromosomes & Cancer|January 26, 2025
Structural Variant Analysis of Complex Karyotype Myelodysplastic Neoplasia Through Optical Genome MappingAndriana Valkama, Sandra Vorimo, Anna Tervasmäki, et al.
Plos Genetics|January 29, 2016
Targeted Next-Generation Sequencing Identifies a Recurrent Mutation in MCPH1 Associating with Hereditary Breast Cancer SusceptibilityTuomo Mantere, Robert Winqvist, Saila Kauppila, et al.
International Journal of Cancer|February 28, 2019
Tumor suppressor MCPH1 regulates gene expression profiles related to malignant conversion and chromosomal assemblyAnna Tervasmäki, Tuomo Mantere, Leila Eshraghi, et al.
Molecular Genetics & Genomic Medicine|September 19, 2020
Evaluating the role of NTHL1 p.Q90* allele in inherited breast cancer predispositionTimo Kumpula, Anna Tervasmäki, Tuomo Mantere, et al.
Familial Cancer|May 19, 2022
Truncating TINF2 p.Tyr312Ter variant and inherited breast cancer susceptibilitySusanna Koivuluoma, Sandra Vorimo, Tiina M Mattila, et al.
Cellular and Molecular Life Sciences : CMLS|April 10, 2024
PALB2-mutated human mammary cells display a broad spectrum of morphological and functional abnormalities induced by increased TGFβ signalingHanna Tuppurainen, Niina Laurila, Marjut Nätynki, et al.
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