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Advances in Experimental Medicine and Biology|May 3, 2022
Sphingolipid Metabolism and Signaling in Endothelial Cell FunctionsLinda Sasset, Annarita Di Lorenzo
The Journal of Pharmacology and Experimental Therapeutics|June 19, 2016
S1P Signaling and De Novo Biosynthesis in Blood Pressure HomeostasisAnna Cantalupo, Annarita Di Lorenzo
International Journal of Molecular Sciences|February 9, 2020
Sphingolipid Signature of Human Feto-Placental Vasculature in PreeclampsiaIlaria Del Gaudio, Linda Sasset, Annarita Di Lorenzo, et al.
Trends in Endocrinology and Metabolism: TEM|August 27, 2016
Sphingolipid De Novo Biosynthesis: A Rheostat of Cardiovascular HomeostasisLinda Sasset, Yi Zhang, Teresa M Dunn, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 23, 2009
Akt1 is critical for acute inflammation and histamine-mediated vascular leakageAnnarita Di Lorenzo, Carlos Fernández-Hernando, Giuseppe Cirino, et al.
The American Journal of Pathology|November 23, 2013
IgE receptor-mediated mast-cell renin releaseSilvia Aldi, Pablo A Robador, Kengo Tomita, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|June 30, 2010
Telmisartan regresses left ventricular hypertrophy in caveolin-1-deficient miceMarta H Krieger, Annarita Di Lorenzo, Christine Teutsch, et al.
Journal of Vascular Research|September 16, 2020
Early Morphofunctional Changes in AngII-Infused Mice Contribute to Regional Onset of Aortic Aneurysm and DissectionLydia Aslanidou, Bram Trachet, Linda Sasset, et al.
The Journal of Pharmacology and Experimental Therapeutics|August 7, 2004
Nitric oxide (NO)-releasing naproxen (HCT-3012 [(S)-6-methoxy-alpha-methyl-2-naphthaleneacetic Acid 4-(nitrooxy)butyl ester]) interactions with aspirin in gastric mucosa of arthritic rats reveal a role for aspirin-triggered lipoxin, prostaglandins, and NO in gastric protectionStefano Fiorucci, Annarita Di Lorenzo, Barbara Renga, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|September 19, 2009
Absence of Akt1 reduces vascular smooth muscle cell migration and survival and induces features of plaque vulnerability and cardiac dysfunction during atherosclerosisCarlos Fernández-Hernando, Levente József, Deborah Jenkins, et al.
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