Showing results (1-10 of 9) with videos related to
Sort By:
Pageof 1
Cancer Research|August 5, 2008
Metastasis suppressor function of tumor necrosis factor-related apoptosis-inducing ligand-R in mice: implications for TRAIL-based therapy in humans?Anne Grosse-Wilde, Christopher J KempMolecular Cancer Research : MCR|July 23, 2008
Transforming growth factor-beta-mediated tumor necrosis factor-related apoptosis-inducing ligand expression and apoptosis in hepatoma cells requires functional cooperation between Smad proteins and activator protein-1Kerstin Herzer, Anne Grosse-Wilde, Peter H Krammer, et al.The EMBO Journal|August 29, 2002
Caspase-10 is recruited to and activated at the native TRAIL and CD95 death-inducing signalling complexes in a FADD-dependent manner but can not functionally substitute caspase-8Martin R Sprick, Eva Rieser, Heiko Stahl, et al.Hepatology (Baltimore, Md.)|June 18, 2005
Transforming growth factor beta can mediate apoptosis via the expression of TRAIL in human hepatoma cellsKerstin Herzer, Tom M Ganten, Henning Schulze-Bergkamen, et al.Journal of Immunology (Baltimore, Md. : 1950)|February 8, 2003
TNF-related apoptosis-inducing ligand mediates tumoricidal activity of human monocytes stimulated by Newcastle disease virusBirgit Washburn, Markus A Weigand, Anne Grosse-Wilde, et al.Plos One|May 29, 2015
Stemness of the hybrid Epithelial/Mesenchymal State in Breast Cancer and Its Association with Poor SurvivalAnne Grosse-Wilde, Aymeric Fouquier d'Hérouël, Ellie McIntosh, et al.Oncotarget|May 8, 2018
Loss of inter-cellular cooperation by complete epithelial-mesenchymal transition supports favorable outcomes in basal breast cancer patientsAnne Grosse-Wilde, Rolf E Kuestner, Stephanie M Skelton, et al.The Journal of Clinical Investigation|December 15, 2007
TRAIL-R deficiency in mice enhances lymph node metastasis without affecting primary tumor developmentAnne Grosse-Wilde, Oksana Voloshanenko, S Lawrence Bailey, et al.Cancer Cell|April 7, 2015
Cancer cell-autonomous TRAIL-R signaling promotes KRAS-driven cancer progression, invasion, and metastasisSilvia von Karstedt, Annalisa Conti, Max Nobis, et al.Pageof 1