Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Anne Joutel

Showing results (51-60 of 74) with videos related to

Pageof 8
Sort By:
Acta Neuropathologica|May 5, 2018
CADASIL brain vessels show a HTRA1 loss-of-function profileAndreas Zellner, Eva Scharrer, Thomas Arzberger, et al.
Hypertension (Dallas, Tex. : 1979)|April 27, 2011
Notch3 is essential for regulation of the renal vascular toneNada Boulos, Frank Helle, Jean-Claude Dussaule, et al.
Brain : a Journal of Neurology|January 29, 2009
Congruence between NOTCH3 mutations and GOM in 131 CADASIL patientsSaara Tikka, Kati Mykkänen, Marie-Magdeleine Ruchoux, et al.
Brain : a Journal of Neurology|May 8, 2013
Abnormal recruitment of extracellular matrix proteins by excess Notch3 ECD: a new pathomechanism in CADASILMarie Monet-Leprêtre, Iman Haddad, Céline Baron-Menguy, et al.
The Journal of Clinical Investigation|January 15, 2010
Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel diseaseAnne Joutel, Marie Monet-Leprêtre, Claudia Gosele, et al.
Elife|August 2, 2016
Mechanistic insights into a TIMP3-sensitive pathway constitutively engaged in the regulation of cerebral hemodynamicsCarmen Capone, Fabrice Dabertrand, Celine Baron-Menguy, et al.
The Journal of Clinical Investigation|February 22, 2024
Protein aggregates containing wild-type and mutant NOTCH3 are major drivers of arterial pathology in CADASILNicolas Dupré, Florian Gueniot, Valérie Domenga-Denier, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 4, 2015
Potassium channelopathy-like defect underlies early-stage cerebrovascular dysfunction in a genetic model of small vessel diseaseFabrice Dabertrand, Christel Krøigaard, Adrian D Bonev, et al.
Genes & Development|November 17, 2004
Notch3 is required for arterial identity and maturation of vascular smooth muscle cellsValérie Domenga, Peggy Fardoux, Pierre Lacombe, et al.
Journal of Neurology|September 27, 2020
A NOTCH3 homozygous nonsense mutation in familial Sneddon syndrome with pediatric strokeElli Katharine Greisenegger, Sara Llufriu, Angel Chamorro, et al.
Pageof 8

Showing results (51-60 of 74) with videos related to

Sort By:
Pageof 8
Acta Neuropathologica|May 5, 2018
CADASIL brain vessels show a HTRA1 loss-of-function profileAndreas Zellner, Eva Scharrer, Thomas Arzberger, et al.
Hypertension (Dallas, Tex. : 1979)|April 27, 2011
Notch3 is essential for regulation of the renal vascular toneNada Boulos, Frank Helle, Jean-Claude Dussaule, et al.
Brain : a Journal of Neurology|January 29, 2009
Congruence between NOTCH3 mutations and GOM in 131 CADASIL patientsSaara Tikka, Kati Mykkänen, Marie-Magdeleine Ruchoux, et al.
Brain : a Journal of Neurology|May 8, 2013
Abnormal recruitment of extracellular matrix proteins by excess Notch3 ECD: a new pathomechanism in CADASILMarie Monet-Leprêtre, Iman Haddad, Céline Baron-Menguy, et al.
The Journal of Clinical Investigation|January 15, 2010
Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel diseaseAnne Joutel, Marie Monet-Leprêtre, Claudia Gosele, et al.
Elife|August 2, 2016
Mechanistic insights into a TIMP3-sensitive pathway constitutively engaged in the regulation of cerebral hemodynamicsCarmen Capone, Fabrice Dabertrand, Celine Baron-Menguy, et al.
The Journal of Clinical Investigation|February 22, 2024
Protein aggregates containing wild-type and mutant NOTCH3 are major drivers of arterial pathology in CADASILNicolas Dupré, Florian Gueniot, Valérie Domenga-Denier, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 4, 2015
Potassium channelopathy-like defect underlies early-stage cerebrovascular dysfunction in a genetic model of small vessel diseaseFabrice Dabertrand, Christel Krøigaard, Adrian D Bonev, et al.
Genes & Development|November 17, 2004
Notch3 is required for arterial identity and maturation of vascular smooth muscle cellsValérie Domenga, Peggy Fardoux, Pierre Lacombe, et al.
Journal of Neurology|September 27, 2020
A NOTCH3 homozygous nonsense mutation in familial Sneddon syndrome with pediatric strokeElli Katharine Greisenegger, Sara Llufriu, Angel Chamorro, et al.
Pageof 8