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Plos Biology|September 5, 2024
Centrosome amplification primes ovarian cancer cells for apoptosis and potentiates the response to chemotherapyFrances Edwards, Giulia Fantozzi, Anthony Y Simon, et al.
Trials|June 9, 2019
Intravenous immunoglobulin and rituximab versus placebo treatment of antibody-associated psychosis: study protocol of a randomised phase IIa double-blinded placebo-controlled trial (SINAPPS2)Belinda Lennox, Ksenija Yeeles, Peter B Jones, et al.
The American Journal of Pathology|August 4, 2018
Medullary Breast Carcinoma, a Triple-Negative Breast Cancer Associated with BCLG OverexpressionPierre Romero, Vanessa Benhamo, Gabrielle Deniziaut, et al.
Plos One|October 12, 2012
Non-sentinel lymph node metastasis prediction in breast cancer with metastatic sentinel lymph node: impact of molecular subtypes classificationFabien Reyal, Catherine Belichard, Roman Rouzier, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 29, 2018
Prognostic Impact of Residual HPV ctDNA Detection after Chemoradiotherapy for Anal Squamous Cell CarcinomaLuc Cabel, Emmanuelle Jeannot, Ivan Bieche, et al.
NPJ Breast Cancer|March 2, 2019
ESR1 mutations in metastatic lobular breast cancer patientsChristine Desmedt, Julien Pingitore, Françoise Rothé, et al.
Breast Cancer Research : BCR|April 10, 2007
Identification of typical medullary breast carcinoma as a genomic sub-group of basal-like carcinomas, a heterogeneous new molecular entityAnne Vincent-Salomon, Nadège Gruel, Carlo Lucchesi, et al.
Oncoimmunology|October 27, 2023
CD39+ conventional CD4+ T cells with exhaustion traits and cytotoxic potential infiltrate tumors and expand upon CTLA-4 blockadeSabrina N Bossio, Carolina Abrate, Jimena Tosello Boari, et al.
Cancer Research|July 21, 2022
Lipid-Associated Macrophages Are Induced by Cancer-Associated Fibroblasts and Mediate Immune Suppression in Breast CancerEleonora Timperi, Paul Gueguen, Martina Molgora, et al.
Nature Communications|August 22, 2014
Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivationPietro Mesirca, Jacqueline Alig, Angelo G Torrente, et al.
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