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Journal of Molecular Biology|September 7, 2021
The Nt17 Domain and its Helical Conformation Regulate the Aggregation, Cellular Properties and Neurotoxicity of Mutant Huntingtin Exon 1Sophie Vieweg, Anne-Laure Mahul-Mellier, Francesco S Ruggeri, et al.
ACS Chemical Biology|June 30, 2016
Semisynthetic and in Vitro Phosphorylation of Alpha-Synuclein at Y39 Promotes Functional Partly Helical Membrane-Bound States Resembling Those Induced by PD MutationsIgor Dikiy, Bruno Fauvet, Ana Jovičić, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 18, 2019
α-Synuclein O-GlcNAcylation alters aggregation and toxicity, revealing certain residues as potential inhibitors of Parkinson's diseasePaul M Levine, Ana Galesic, Aaron T Balana, et al.
Oncotarget|June 11, 2016
Synergy between histone deacetylase inhibitors and DNA-damaging agents is mediated by histone deacetylase 2 in colorectal cancerSamer Alzoubi, Leigh Brody, Sunniyat Rahman, et al.
Biochemical Society Transactions|January 16, 2009
Alix and ALG-2 make a link between endosomes and neuronal deathAnne-Laure Mahul-Mellier, Flavie Strappazzon, Christine Chatellard-Causse, et al.
Nature Communications|November 13, 2021
Nuclear and cytoplasmic huntingtin inclusions exhibit distinct biochemical composition, interactome and ultrastructural propertiesNathan Riguet, Anne-Laure Mahul-Mellier, Niran Maharjan, et al.
The Journal of Biological Chemistry|October 22, 2008
Alix and ALG-2 are involved in tumor necrosis factor receptor 1-induced cell deathAnne-Laure Mahul-Mellier, Flavie Strappazzon, Anne Petiot, et al.
Human Molecular Genetics|January 14, 2014
c-Abl phosphorylates α-synuclein and regulates its degradation: implication for α-synuclein clearance and contribution to the pathogenesis of Parkinson's diseaseAnne-Laure Mahul-Mellier, Bruno Fauvet, Amanda Gysbers, et al.
The Journal of Biological Chemistry|June 18, 2014
The H50Q mutation enhances α-synuclein aggregation, secretion, and toxicityOssama Khalaf, Bruno Fauvet, Abid Oueslati, et al.
Science Advances|April 29, 2022
A NAC domain mutation (E83Q) unlocks the pathogenicity of human alpha-synuclein and recapitulates its pathological diversitySenthil T Kumar, Anne-Laure Mahul-Mellier, Ramanath Narayana Hegde, et al.
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