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Molecular Endocrinology (Baltimore, Md.)|March 16, 2004
Insight into mutation-induced activation of the luteinizing hormone receptor: molecular simulations predict the functional behavior of engineered mutants at M398Francesca Fanelli, Miriam Verhoef-Post, Marianna Timmerman, et al.Experimental Cell Research|June 24, 2004
Telomerase activity level, but not hTERT mRNA and hTR level, regulates telomere length in telomerase-reconstituted primary fibroblastsSusan J J Swiggers, H Arno J Nibbeling, Annelieke Zeilemaker, et al.Blood Advances|December 18, 2024
Double mutant DNMT3A AML: a unique subtype experiencing increased DNA damage and poor prognosisEmma L Boertjes, Sanne Massaar, Annelieke Zeilemaker, et al.Blood|June 12, 2010
Acquired mutations in the genes encoding IDH1 and IDH2 both are recurrent aberrations in acute myeloid leukemia: prevalence and prognostic valueSaman Abbas, Sanne Lugthart, François G Kavelaars, et al.Blood|April 12, 2012
Mutant DNMT3A: a marker of poor prognosis in acute myeloid leukemiaAna Flávia Tibúrcio Ribeiro, Marta Pratcorona, Claudia Erpelinck-Verschueren, et al.Blood|November 9, 2014
Mutational spectrum of myeloid malignancies with inv(3)/t(3;3) reveals a predominant involvement of RAS/RTK signaling pathwaysStefan Gröschel, Mathijs A Sanders, Remco Hoogenboezem, et al.Haematologica|January 21, 2014
Integrated genome-wide genotyping and gene expression profiling reveals BCL11B as a putative oncogene in acute myeloid leukemia with 14q32 aberrationsSaman Abbas, Mathijs A Sanders, Annelieke Zeilemaker, et al.Blood|February 14, 2019
Altered NFE2 activity predisposes to leukemic transformation and myelosarcoma with AML-specific aberrationsJonas Samuel Jutzi, Titiksha Basu, Maximilian Pellmann, et al.Blood|July 28, 2018
MBD4 guards against methylation damage and germ line deficiency predisposes to clonal hematopoiesis and early-onset AMLMathijs A Sanders, Edward Chew, Christoffer Flensburg, et al.The New England Journal of Medicine|March 31, 2018
Molecular Minimal Residual Disease in Acute Myeloid LeukemiaMojca Jongen-Lavrencic, Tim Grob, Diana Hanekamp, et al.Pageof 1