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Anthony A Azenabor

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Comparative Immunology, Microbiology and Infectious Diseases|September 29, 2009
Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophagesAnthony A Azenabor, Jenniffer York
Medical Microbiology and Immunology|May 9, 2003
Effective macrophage redox defense against Chlamydia pneumoniae depends on L-type Ca2+ channel activationAnthony A Azenabor, Aziz U Chaudhry
Biological Chemistry|February 24, 2004
Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling eventsAnthony A Azenabor, Godwin Job, Shoua Yang
International Journal of Medical Sciences|September 13, 2007
Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelaeAnthony A Azenabor, Patrick Kennedy, Salvatore Balistreri
Immunobiology|January 27, 2009
Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophagesAnthony A Azenabor, Patrick Kennedy, Jenniffer York
Immunobiology|September 4, 2003
Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibilityAnthony A Azenabor, Aziz U Chaudhry, Shoua Yang
Molecular and Cellular Biochemistry|March 25, 2005
Chlamydia pneumoniae infected macrophages exhibit enhanced plasma membrane fluidity and show increased adherence to endothelial cellsAnthony A Azenabor, Godwin Job, Olanrewaju O Adedokun
Medical Microbiology and Immunology|June 16, 2004
Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genesAnthony A Azenabor, Shoua Yang, Godwin Job, et al.
Immunobiology|May 24, 2006
Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: evidence of the effect of redox balance on host-pathogen relationshipAnthony A Azenabor, Kamaldeen Muili, Jane-Francis Akoachere, et al.
Immunobiology|August 2, 2011
Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cellsAnthony A Azenabor, Jenniffer Cintrón-Cuevas, Heather Schmitt, et al.
Pageof 2

Showing results (1-10 of 15) with videos related to

Sort By:
Pageof 2
Comparative Immunology, Microbiology and Infectious Diseases|September 29, 2009
Chlamydia trachomatis evokes a relative anti-inflammatory response in a free Ca2+ dependent manner in human macrophagesAnthony A Azenabor, Jenniffer York
Medical Microbiology and Immunology|May 9, 2003
Effective macrophage redox defense against Chlamydia pneumoniae depends on L-type Ca2+ channel activationAnthony A Azenabor, Aziz U Chaudhry
Biological Chemistry|February 24, 2004
Induction of lipoprotein lipase gene expression in Chlamydia pneumoniae-infected macrophages is dependent on Ca2+ signaling eventsAnthony A Azenabor, Godwin Job, Shoua Yang
International Journal of Medical Sciences|September 13, 2007
Chlamydia trachomatis infection of human trophoblast alters estrogen and progesterone biosynthesis: an insight into role of infection in pregnancy sequelaeAnthony A Azenabor, Patrick Kennedy, Salvatore Balistreri
Immunobiology|January 27, 2009
Free intracellular Ca2+ regulates bacterial lipopolysaccharide induction of iNOS in human macrophagesAnthony A Azenabor, Patrick Kennedy, Jenniffer York
Immunobiology|September 4, 2003
Macrophage L-type Ca2+ channel antagonists alter Chlamydia pneumoniae MOMP and HSP-60 mRNA gene expression, and improve antibiotic susceptibilityAnthony A Azenabor, Aziz U Chaudhry, Shoua Yang
Molecular and Cellular Biochemistry|March 25, 2005
Chlamydia pneumoniae infected macrophages exhibit enhanced plasma membrane fluidity and show increased adherence to endothelial cellsAnthony A Azenabor, Godwin Job, Olanrewaju O Adedokun
Medical Microbiology and Immunology|June 16, 2004
Elicitation of reactive oxygen species in Chlamydia pneumoniae-stimulated macrophages: a Ca2+-dependent process involving simultaneous activation of NADPH oxidase and cytochrome oxidase genesAnthony A Azenabor, Shoua Yang, Godwin Job, et al.
Immunobiology|May 24, 2006
Macrophage antioxidant enzymes regulate Chlamydia pneumoniae chronicity: evidence of the effect of redox balance on host-pathogen relationshipAnthony A Azenabor, Kamaldeen Muili, Jane-Francis Akoachere, et al.
Immunobiology|August 2, 2011
Chlamydia trachomatis induces anti-inflammatory effect in human macrophages by attenuation of immune mediators in Jurkat T-cellsAnthony A Azenabor, Jenniffer Cintrón-Cuevas, Heather Schmitt, et al.
Pageof 2