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Physiology (Bethesda, Md.)|September 24, 2004
The nuclear envelope and human diseaseAntoine Muchir, Howard J WormanMethods in Enzymology|January 23, 2016
Targeting Mitogen-Activated Protein Kinase Signaling in Mouse Models of Cardiomyopathy Caused by Lamin A/C Gene MutationsAntoine Muchir, Howard J WormanDevelopmental Cell|September 14, 2010
Signaling defects and the nuclear envelope in progeriaAntoine Muchir, Howard J WormanCurrent Opinion in Neurology|August 29, 2019
Emery-Dreifuss muscular dystrophy: focal point nuclear envelopeAntoine Muchir, Howard J WormanCurrent Neurology and Neuroscience Reports|January 16, 2007
Emery-Dreifuss muscular dystrophyAntoine Muchir, Howard J WormanBiochimica Et Biophysica Acta|November 22, 2008
Reduced expression of A-type lamins and emerin activates extracellular signal-regulated kinase in cultured cellsAntoine Muchir, Wei Wu, Howard J WormanTrends in Cardiovascular Medicine|February 2, 2012
Mitogen-activated protein kinase inhibitor regulation of heart function and fibrosis in cardiomyopathy caused by lamin A/C gene mutationAntoine Muchir, Wei Wu, Howard J WormanThe Journal of Clinical Investigation|July 10, 2009
Laminopathies and the long strange trip from basic cell biology to therapyHoward J Worman, Loren G Fong, Antoine Muchir, et al.Biochemical and Biophysical Research Communications|September 15, 2014
Mitogen-activated protein kinase kinase 1/2 inhibition and angiotensin II converting inhibition in mice with cardiomyopathy caused by lamin A/C gene mutationAntoine Muchir, Wei Wu, Fusako Sera, et al.Disease Models & Mechanisms|August 4, 2011
LMNA cardiomyopathy: cell biology and genetics meet clinical medicineJonathan T Lu, Antoine Muchir, Peter L Nagy, et al.Pageof 19