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Human Molecular Genetics|September 2, 2024
NEK1 haploinsufficiency worsens DNA damage, but not defective ciliogenesis, in C9ORF72 patient-derived iPSC-motoneuronsSerena Santangelo, Sabrina Invernizzi, Marta Nice Sorce, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|December 15, 2023
Analysis of normal C9orf72 repeat length as possible disease modifier in amyotrophic lateral sclerosisSilvia Peverelli, Alberto Brusati, Valeria Casiraghi, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|July 6, 2022
Expanding the phenotype of TARDBP mutation in a Tunisian family with clinical phenotype heterogeneityImen Kacem, Ikram Sghaier, Nicola Ticozzi, et al.
Journal of the Neurological Sciences|September 6, 2024
Quantification of serum TDP-43 and neurofilament light chain in patients with amyotrophic lateral sclerosis stratified by UNC13A genotypeValeria Casiraghi, Ilaria Milone, Alberto Brusati, et al.
Neurological Sciences : Official Journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology|March 24, 2019
PON1 is a disease modifier gene in amyotrophic lateral sclerosis: association of the Q192R polymorphism with bulbar onset and reduced survivalFederico Verde, Cinzia Tiloca, Claudia Morelli, et al.
Neurobiology of Disease|August 23, 2020
Chronic stress induces formation of stress granules and pathological TDP-43 aggregates in human ALS fibroblasts and iPSC-motoneuronsAntonia Ratti, Valentina Gumina, Paola Lenzi, et al.
Acta Neuropathologica Communications|May 7, 2016
Gene-specific mitochondria dysfunctions in human TARDBP and C9ORF72 fibroblastsElisa Onesto, Claudia Colombrita, Valentina Gumina, et al.
Frontiers in Aging Neuroscience|October 17, 2022
TARDBP mutations in a cohort of Italian patients with Parkinson's disease and atypical parkinsonismsCinzia Tiloca, Stefano Goldwurm, Narghes Calcagno, et al.
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