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Cold Spring Harbor Perspectives in Biology|April 23, 2017
Do Memory CD4 T Cells Keep Their Cell-Type Programming: Plasticity versus Fate Commitment? T-Cell Heterogeneity, Plasticity, and Selection in HumansFederica Sallusto, Antonino Cassotta, Daniel Hoces, et al.Journal of Leukocyte Biology|August 28, 2015
Regulation and trafficking of the HLA-E molecules during monocyte-macrophage differentiationGiorgio Camilli, Antonino Cassotta, Simone Battella, et al.European Journal of Immunology|December 8, 2022
Clonal composition and persistence of antigen-specific circulating T follicular helper cellsMengyun Hu, Samuele Notarbartolo, Mathilde Foglierini, et al.Nature Immunology|June 27, 2022
Clonal structure, stability and dynamics of human memory B cells and circulating plasmablastsGanesh E Phad, Dora Pinto, Mathilde Foglierini, et al.The Journal of Experimental Medicine|July 10, 2020
Deciphering and predicting CD4+ T cell immunodominance of influenza virus hemagglutininAntonino Cassotta, Philipp Paparoditis, Roger Geiger, et al.Science (New York, N.Y.)|December 6, 2014
T cell immunity. Functional heterogeneity of human memory CD4⁺ T cell clones primed by pathogens or vaccinesSimone Becattini, Daniela Latorre, Federico Mele, et al.European Journal of Immunology|November 23, 2020
Broadly reactive human CD4+ T cells against Enterobacteriaceae are found in the naïve repertoire and are clonally expanded in the memory repertoireAntonino Cassotta, Jérémie D Goldstein, Greta Durini, et al.European Journal of Immunology|December 12, 2024
Dual Activation-Induced Marker Combinations Efficiently Identify and Discern Antigen-Specific and Bystander-Activated Human CD4+ T CellsMaria Grazia Ceraolo, Maristella Leccese, Antonino Cassotta, et al.The Journal of Experimental Medicine|October 15, 2025
FCRL3 is an immunoregulatory receptor that restrains the activation of human memory T lymphocytesNiccolò Bianchi, Elena Foli, Mehrpouya Mostanfar, et al.Science Signaling|September 23, 2025
Adenosine 2A receptor-dependent activation of AMPK represses TH17 cell pathogenicity through epigenetic and metabolic reprogrammingGina Papadopoulou, Dimitrios Valakos, Ioanna Polydouri, et al.Pageof 3