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Scientific Reports|May 17, 2022
Heat shock protein Grp78/BiP/HspA5 binds directly to TDP-43 and mitigates toxicity associated with disease pathologyLiberty François-Moutal, David Donald Scott, Andrew J Ambrose, et al.Elife|December 15, 2020
Therapeutic genetic variation revealed in diverse Hsp104 homologsZachary M March, Katelyn Sweeney, Hanna Kim, et al.Alzheimer'S Research & Therapy|June 30, 2019
Spermidine/spermine-N1-acetyltransferase ablation impacts tauopathy-induced polyamine stress responseLeslie A Sandusky-Beltran, Andrii Kovalenko, Chao Ma, et al.Cell|August 11, 2021
TDP-43 condensation properties specify its RNA-binding and regulatory repertoireMartina Hallegger, Anob M Chakrabarti, Flora C Y Lee, et al.Proceedings of the National Academy of Sciences of the United States of America|May 16, 2008
Direct and selective elimination of specific prions and amyloids by 4,5-dianilinophthalimide and analogsHuan Wang, Martin L Duennwald, Blake E Roberts, et al.Cell Reports|August 22, 2019
Mining Disaggregase Sequence Space to Safely Counter TDP-43, FUS, and α-Synuclein ProteotoxicityAmber Tariq, JiaBei Lin, Meredith E Jackrel, et al.Nature|November 23, 2018
TDP-43 and RNA form amyloid-like myo-granules in regenerating muscleThomas O Vogler, Joshua R Wheeler, Eric D Nguyen, et al.The Journal of Clinical Investigation|February 11, 2014
ALS-associated mutation FUS-R521C causes DNA damage and RNA splicing defectsHaiyan Qiu, Sebum Lee, Yulei Shang, et al.Journal of Molecular Biology|January 23, 2023
Phase Separation in Biology and Disease; Current Perspectives and Open QuestionsSteven Boeynaems, Shasha Chong, Jörg Gsponer, et al.Acta Neuropathologica|March 31, 2023
C-terminal frameshift variant of TDP-43 with pronounced aggregation-propensity causes rimmed vacuole myopathy but not ALS/FTDPedro Ervilha Pereira, Nika Schuermans, Antoon Meylemans, et al.Pageof 21