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Neurobiology of Disease|September 11, 2012
Defects in neuromuscular junction remodelling in the Smn(2B/-) mouse model of spinal muscular atrophyLyndsay M Murray, Ariane Beauvais, Kunal Bhanot, et al.Human Molecular Genetics|January 26, 2010
Rho-kinase inactivation prolongs survival of an intermediate SMA mouse modelMélissa Bowerman, Ariane Beauvais, Carrie L Anderson, et al.Journal of Neuromuscular Diseases|April 26, 2020
Blood Flow to the Spleen is Altered in a Mouse Model of Spinal Muscular AtrophyMarc-Olivier Deguise, Ariane Beauvais, Bernard L Schneider, et al.Eneuro|May 18, 2019
Influence of Temperature on Motor Behaviors in Newborn Opossums (Monodelphis domestica): An In Vitro StudyEdith Corriveau-Parenteau, Ariane Beauvais, Annie Angers, et al.Plos One|July 2, 2014
The Smn-independent beneficial effects of trichostatin A on an intermediate mouse model of spinal muscular atrophyHong Liu, Armin Yazdani, Lyndsay M Murray, et al.Neuromuscular Disorders : NMD|November 11, 2011
A critical smn threshold in mice dictates onset of an intermediate spinal muscular atrophy phenotype associated with a distinct neuromuscular junction pathologyMélissa Bowerman, Lyndsay M Murray, Ariane Beauvais, et al.Human Molecular Genetics|December 11, 2023
Long term peripheral AAV9-SMN gene therapy promotes survival in a mouse model of spinal muscular atrophyAoife Reilly, Rebecca Yaworski, Ariane Beauvais, et al.Annals of Clinical and Translational Neurology|October 15, 2019
Low fat diets increase survival of a mouse model of spinal muscular atrophyMarc-Olivier Deguise, Lucia Chehade, Alexandra Tierney, et al.Developmental Neurobiology|September 24, 2010
Smn deficiency causes neuritogenesis and neurogenesis defects in the retinal neurons of a mouse model of spinal muscular atrophyHong Liu, Ariane Beauvais, Adam N Baker, et al.Plos One|May 17, 2011
The proteolipid protein promoter drives expression outside of the oligodendrocyte lineage during embryonic and early postnatal developmentJohn-Paul Michalski, Carrie Anderson, Ariane Beauvais, et al.Pageof 4