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Scientific Reports|August 27, 2017
The E-cadherin/AmotL2 complex organizes actin filaments required for epithelial hexagonal packing and blastocyst hatchingSebastian Hildebrand, Sara Hultin, Aravindh Subramani, et al.Plos Biology|January 9, 2009
Collective cell migration drives morphogenesis of the kidney nephronAleksandr Vasilyev, Yan Liu, Sudha Mudumana, et al.Blood|October 1, 2008
The Amot/Patj/Syx signaling complex spatially controls RhoA GTPase activity in migrating endothelial cellsMira Ernkvist, Nathalie Luna Persson, Stéphane Audebert, et al.Journal of the American Society of Nephrology : JASN|May 16, 2009
Amino acid transporter LAT3 is required for podocyte development and functionYuji Sekine, Yukino Nishibori, Yoshihiro Akimoto, et al.Genes & Development|August 19, 2007
Angiomotin regulates endothelial cell migration during embryonic angiogenesisKarin Aase, Mira Ernkvist, Lwaki Ebarasi, et al.Nature Communications|May 9, 2014
AmotL2 links VE-cadherin to contractile actin fibres necessary for aortic lumen expansionSara Hultin, Yujuan Zheng, Mahdi Mojallal, et al.Nature Genetics|September 4, 2007
Modulation of morphogenesis by noncanonical Wnt signaling requires ATF/CREB family-mediated transcriptional activation of TGFbeta2Wenlai Zhou, Lizhu Lin, Arindam Majumdar, et al.Nature Communications|April 12, 2013
VE-PTP regulates VEGFR2 activity in stalk cells to establish endothelial cell polarity and lumen formationMakoto Hayashi, Arindam Majumdar, Xiujuan Li, et al.JCI Insight|March 10, 2025
Leveraging complementary multi-omics data integration methods for mechanistic insights in kidney diseasesFadhl Alakwaa, Vivek Das, Arindam Majumdar, et al.American Journal of Human Genetics|January 6, 2015
Defects of CRB2 cause steroid-resistant nephrotic syndromeLwaki Ebarasi, Shazia Ashraf, Agnieszka Bierzynska, et al.Pageof 3