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Arleen B Rifkind

Showing results (1-10 of 14) with videos related to

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Drug Metabolism Reviews|May 11, 2006
CYP1A in TCDD toxicity and in physiology-with particular reference to CYP dependent arachidonic acid metabolism and other endogenous substratesArleen B Rifkind
Nature|August 31, 2012
Competing interests: Expanding rapidlyArleen B Rifkind
Plos One|December 15, 2020
Organ-specific effects on glycolysis by the dioxin-activated aryl hydrocarbon receptorSilvia Diani-Moore, Tiago Marques Pedro, Arleen B Rifkind
Archives of Biochemistry and Biophysics|October 26, 2007
Mitochondrial P450-dependent arachidonic acid metabolism by TCDD-induced hepatic CYP1A5; conversion of EETs to DHETs by mitochondrial soluble epoxide hydrolaseErin M Labitzke, Silvia Diani-Moore, Arleen B Rifkind
Drug Metabolism and Disposition: the Biological Fate of Chemicals|May 23, 2006
Induction of CYP1A and cyp2-mediated arachidonic acid epoxygenation and suppression of 20-hydroxyeicosatetraenoic acid by imidazole derivatives including the aromatase inhibitor vorozoleSilvia Diani-Moore, Fotini Papachristou, Erin Labitzke, et al.
The Journal of Biological Chemistry|June 18, 2013
Aryl hydrocarbon receptor activation by dioxin targets phosphoenolpyruvate carboxykinase (PEPCK) for ADP-ribosylation via 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-inducible poly(ADP-ribose) polymerase (TiPARP)Silvia Diani-Moore, Sheng Zhang, Payal Ram, et al.
Drug Metabolism and Disposition: the Biological Fate of Chemicals|December 7, 2013
Increases in levels of epoxyeicosatrienoic and dihydroxyeicosatrienoic acids (EETs and DHETs) in liver and heart in vivo by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and in hepatic EET:DHET ratios by cotreatment with TCDD and the soluble epoxide hydrolase inhibitor AUDASilvia Diani-Moore, Yuliang Ma, Steven S Gross, et al.
Scientific Reports|May 25, 2017
NAD<sup>+</sup> loss, a new player in AhR biology: prevention of thymus atrophy and hepatosteatosis by NAD<sup>+</sup> repletionSilvia Diani-Moore, Jenny Shoots, Rubi Singh, et al.
The Journal of Biological Chemistry|June 19, 2021
Roles of the ubiquitin ligase CUL4B and ADP-ribosyltransferase TiPARP in TCDD-induced nuclear export and proteasomal degradation of the transcription factor AHRMercedes Paz Rijo, Silvia Diani-Moore, Chenyi Yang, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|December 7, 2005
Sunlight generates multiple tryptophan photoproducts eliciting high efficacy CYP1A induction in chick hepatocytes and in vivoSilvia Diani-Moore, Erin Labitzke, Richard Brown, et al.
Pageof 2

Showing results (1-10 of 14) with videos related to

Sort By:
Pageof 2
Drug Metabolism Reviews|May 11, 2006
CYP1A in TCDD toxicity and in physiology-with particular reference to CYP dependent arachidonic acid metabolism and other endogenous substratesArleen B Rifkind
Nature|August 31, 2012
Competing interests: Expanding rapidlyArleen B Rifkind
Plos One|December 15, 2020
Organ-specific effects on glycolysis by the dioxin-activated aryl hydrocarbon receptorSilvia Diani-Moore, Tiago Marques Pedro, Arleen B Rifkind
Archives of Biochemistry and Biophysics|October 26, 2007
Mitochondrial P450-dependent arachidonic acid metabolism by TCDD-induced hepatic CYP1A5; conversion of EETs to DHETs by mitochondrial soluble epoxide hydrolaseErin M Labitzke, Silvia Diani-Moore, Arleen B Rifkind
Drug Metabolism and Disposition: the Biological Fate of Chemicals|May 23, 2006
Induction of CYP1A and cyp2-mediated arachidonic acid epoxygenation and suppression of 20-hydroxyeicosatetraenoic acid by imidazole derivatives including the aromatase inhibitor vorozoleSilvia Diani-Moore, Fotini Papachristou, Erin Labitzke, et al.
The Journal of Biological Chemistry|June 18, 2013
Aryl hydrocarbon receptor activation by dioxin targets phosphoenolpyruvate carboxykinase (PEPCK) for ADP-ribosylation via 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-inducible poly(ADP-ribose) polymerase (TiPARP)Silvia Diani-Moore, Sheng Zhang, Payal Ram, et al.
Drug Metabolism and Disposition: the Biological Fate of Chemicals|December 7, 2013
Increases in levels of epoxyeicosatrienoic and dihydroxyeicosatrienoic acids (EETs and DHETs) in liver and heart in vivo by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and in hepatic EET:DHET ratios by cotreatment with TCDD and the soluble epoxide hydrolase inhibitor AUDASilvia Diani-Moore, Yuliang Ma, Steven S Gross, et al.
Scientific Reports|May 25, 2017
NAD<sup>+</sup> loss, a new player in AhR biology: prevention of thymus atrophy and hepatosteatosis by NAD<sup>+</sup> repletionSilvia Diani-Moore, Jenny Shoots, Rubi Singh, et al.
The Journal of Biological Chemistry|June 19, 2021
Roles of the ubiquitin ligase CUL4B and ADP-ribosyltransferase TiPARP in TCDD-induced nuclear export and proteasomal degradation of the transcription factor AHRMercedes Paz Rijo, Silvia Diani-Moore, Chenyi Yang, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|December 7, 2005
Sunlight generates multiple tryptophan photoproducts eliciting high efficacy CYP1A induction in chick hepatocytes and in vivoSilvia Diani-Moore, Erin Labitzke, Richard Brown, et al.
Pageof 2