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Nature Structural & Molecular Biology|January 14, 2014
Rapid induction of alternative lengthening of telomeres by depletion of the histone chaperone ASF1Roderick J O'Sullivan, Nausica Arnoult, Daniel H Lackner, et al.
Molecular Biology and Evolution|May 7, 2013
Subfunctionalization via adaptive evolution influenced by genomic context: the case of histone chaperones ASF1a and ASF1bFederico Abascal, Armelle Corpet, Zachary A Gurard-Levin, et al.
Life Science Alliance|March 7, 2023
The TUDOR domain of SMN is an H3K79<sup>me1</sup> histone mark readerOlivier Binda, Aimé Boris Kimenyi Ishimwe, Maxime Galloy, et al.
Elife|May 25, 2023
Interplay between PML NBs and HIRA for H3.3 dynamics following type I interferon stimulusConstance Kleijwegt, Florent Bressac, Coline Seurre, et al.
Molecular Cell|September 20, 2016
Real-Time Tracking of Parental Histones Reveals Their Contribution to Chromatin Integrity Following DNA DamageSalomé Adam, Juliette Dabin, Odile Chevallier, et al.
Neurology|March 30, 2022
Involvement of Type I Interferon Signaling in Muscle Stem Cell Proliferation During DermatomyositisLaure Gallay, Cécile Fermon, Lola Lessard, et al.
Life Science Alliance|November 14, 2022
SMA-linked SMN mutants prevent phase separation properties and SMN interactions with FMRP family membersOlivier Binda, Franceline Juillard, Julia Novion Ducassou, et al.
NAR Molecular Medicine|May 27, 2026
Defective plasticity in dermatomyositis patients muscle stem cells is associated with sustained intrinsic inflammatory signaling and disruption of the histone H3.3 chromatin loading pathwayWilhelm Bouchereau, Linda Chenane, Michèle Weiss-Gayet, et al.
Plos Pathogens|September 21, 2018
Promyelocytic leukemia (PML) nuclear bodies (NBs) induce latent/quiescent HSV-1 genomes chromatinization through a PML NB/Histone H3.3/H3.3 Chaperone AxisCamille Cohen, Armelle Corpet, Simon Roubille, et al.
The EMBO Journal|December 24, 2010
Asf1b, the necessary Asf1 isoform for proliferation, is predictive of outcome in breast cancerArmelle Corpet, Leanne De Koning, Joern Toedling, et al.
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