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Behavioural Brain Research|November 5, 2010
The telomeric part of the human chromosome 21 from Cstb to Prmt2 is not necessary for the locomotor and short-term memory deficits observed in the Tc1 mouse model of Down syndromeArnaud Duchon, Stéphanie Pothion, Véronique Brault, et al.
Human Molecular Genetics|June 27, 2007
Modeling the monosomy for the telomeric part of human chromosome 21 reveals haploinsufficient genes modulating the inflammatory and airway responsesVanessa Besson, Véronique Brault, Arnaud Duchon, et al.
Disease Models & Mechanisms|June 3, 2015
Deletion of the App-Runx1 region in mice models human partial monosomy 21Thomas Arbogast, Matthieu Raveau, Claire Chevalier, et al.
Plos Genetics|June 14, 2012
The App-Runx1 region is critical for birth defects and electrocardiographic dysfunctions observed in a Down syndrome mouse modelMatthieu Raveau, Jacques M Lignon, Valérie Nalesso, et al.
British Journal of Pharmacology|October 26, 2019
Long-lasting correction of in vivo LTP and cognitive deficits of mice modelling Down syndrome with an α5-selective GABAA inverse agonistArnaud Duchon, Agnès Gruart, Christelle Albac, et al.
Molecular Genetics and Metabolism|August 8, 2013
DYRK1A overexpression decreases plasma lecithin:cholesterol acyltransferase activity and apolipoprotein A-I levelsAsma Tlili, Christophe Noll, Sandrine Middendorp, et al.
Neurobiology of Disease|May 5, 2024
Shaping down syndrome brain cognitive and molecular changes due to aging using adult animals from the Ts66Yah murine modelChiara Lanzillotta, Monika Rataj Baniowska, Francesca Prestia, et al.
Scientific Reports|March 10, 2019
Prenatal treatment with EGCG enriched green tea extract rescues GAD67 related developmental and cognitive defects in Down syndrome mouse modelsBenoit Souchet, Arnaud Duchon, Yuchen Gu, et al.
Frontiers in Behavioral Neuroscience|November 6, 2015
Pharmacological correction of excitation/inhibition imbalance in Down syndrome mouse modelsBenoit Souchet, Fayçal Guedj, Zsuza Penke-Verdier, et al.
Disease Models & Mechanisms|November 14, 2022
Ts66Yah, a mouse model of Down syndrome with improved construct and face validityArnaud Duchon, Maria Del Mar Muñiz Moreno, Claire Chevalier, et al.
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