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Blood|December 9, 2022
Contribution of mutant HSC clones to immature and mature cells in MDS and CMML, and variations with AZA therapyAnnatina S Schnegg-Kaufmann, Julie A I Thoms, Golam Sarower Bhuyan, et al.Proceedings of the National Academy of Sciences of the United States of America|April 5, 2016
PDGF-AB and 5-Azacytidine induce conversion of somatic cells into tissue-regenerative multipotent stem cellsVashe Chandrakanthan, Avani Yeola, Jair C Kwan, et al.Nature Biotechnology|June 18, 2021
The RNA Atlas expands the catalog of human non-coding RNAsLucia Lorenzi, Hua-Sheng Chiu, Francisco Avila Cobos, et al.Cell Reports|July 21, 2017
Integrative Genomics Identifies the Molecular Basis of Resistance to Azacitidine Therapy in Myelodysplastic SyndromesAshwin Unnikrishnan, Elli Papaemmanuil, Dominik Beck, et al.Cancer Cell|May 20, 2014
Myelodysplastic syndromes are propagated by rare and distinct human cancer stem cells in vivoPetter S Woll, Una Kjällquist, Onima Chowdhury, et al.Elife|October 2, 2014
Origins and functional consequences of somatic mitochondrial DNA mutations in human cancerYoung Seok Ju, Ludmil B Alexandrov, Moritz Gerstung, et al.Nature Communications|May 13, 2025
Clinical response to azacitidine in MDS is associated with distinct DNA methylation changes in HSPCsJulie A I Thoms, Feng Yan, Henry R Hampton, et al.Pageof 3