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Experimental Cell Research|January 24, 2007
CCN2 is necessary for the function of mouse embryonic fibroblastsLaura Kennedy, Shangxi Liu, Xu Shi-Wen, et al.
Matrix Biology Plus|February 5, 2021
CCN1 expression by fibroblasts is required for bleomycin-induced skin fibrosisKatherine Quesnel, Xu Shi-Wen, James Hutchenreuther, et al.
Clinical Epigenetics|June 3, 2023
Novel insights into systemic sclerosis using a sensitive computational method to analyze whole-genome bisulfite sequencing dataJeffrey C Y Yu, Yixiao Zeng, Kaiqiong Zhao, et al.
Journal of Cellular Physiology|March 29, 2014
Monitoring collagen synthesis in fibroblasts using fluorescently labeled tRNA pairsJiaqi Liu, Macarena Pampillo, Fen Guo, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|August 4, 2024
Matricellular protein CCN1 promotes collagen alignment and scar integrity after myocardial infarctionAnnalara G Fischer, Erin M Elliott, Kenneth R Brittian, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|June 10, 2018
Activation of cancer-associated fibroblasts is required for tumor neovascularization in a murine model of melanomaJames Hutchenreuther, Krista Vincent, Chris Norley, et al.
Cardiovascular Research|October 4, 2024
Cellular communication network factor 2 regulates smooth muscle cell transdifferentiation and lipid accumulation in atherosclerosisQian Xu, Jisheng Sun, Claire M Holden, et al.
Molecular and Cellular Biology|July 1, 2006
Constitutive ALK5-independent c-Jun N-terminal kinase activation contributes to endothelin-1 overexpression in pulmonary fibrosis: evidence of an autocrine endothelin loop operating through the endothelin A and B receptorsXu Shi-Wen, Fernando Rodríguez-Pascual, Santiago Lamas, et al.
Molecular Biology of the Cell|March 30, 2004
Endothelin-1 promotes myofibroblast induction through the ETA receptor via a rac/phosphoinositide 3-kinase/Akt-dependent pathway and is essential for the enhanced contractile phenotype of fibrotic fibroblastsXu Shi-Wen, Yunliang Chen, Christopher P Denton, et al.
The American Journal of Pathology|November 30, 2005
Matrix contraction by dermal fibroblasts requires transforming growth factor-beta/activin-linked kinase 5, heparan sulfate-containing proteoglycans, and MEK/ERK: insights into pathological scarring in chronic fibrotic diseaseYunliang Chen, Xu Shi-Wen, Jonathan van Beek, et al.
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