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American Journal of Physiology. Cell Physiology|August 6, 2003
PKC zeta participates in activation of inflammatory response induced by enteropathogenic E. coliSuzana D Savkovic, Athanasia Koutsouris, Gail Hecht
American Journal of Physiology. Gastrointestinal and Liver Physiology|March 22, 2008
Enteropathogenic E. coli-induced barrier function alteration is not a consequence of host cell apoptosisV K Viswanathan, Andrew Weflen, Athanasia Koutsouris, et al.
Cellular Microbiology|August 29, 2009
The bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by enteropathogenic Escherichia coliAjitha Thanabalasuriar, Athanasia Koutsouris, Andrew Weflen, et al.
American Journal of Physiology. Gastrointestinal and Liver Physiology|June 27, 2009
Enterohemorrhagic Escherichia coli suppresses inflammatory response to cytokines and its own toxinAmy Bellmeyer, Cynthia Cotton, Rajani Kanteti, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|August 30, 2005
Enteropathogenic E. coli disrupts tight junction barrier function and structure in vivoDonnie E Shifflett, Daniel R Clayburgh, Athanasia Koutsouris, et al.
Cellular Microbiology|September 2, 2004
Cytokeratin 18 interacts with the enteropathogenic Escherichia coli secreted protein F (EspF) and is redistributed after infectionV K Viswanathan, Sandra Lukic, Athanasia Koutsouris, et al.
Infection and Immunity|May 25, 2004
Comparative analysis of EspF from enteropathogenic and enterohemorrhagic Escherichia coli in alteration of epithelial barrier functionV K Viswanathan, Athanasia Koutsouris, Sandra Lukic, et al.
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