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Brain Pathology (Zurich, Switzerland)|June 18, 2019
Perturbation of the immune cells and prenatal neurogenesis by the triplication of the Erg gene in mouse models of Down syndromeKeiichi Ishihara, Ryohei Shimizu, Kazuyuki Takata, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 6, 2009
DSCAM deficiency causes loss of pre-inspiratory neuron synchroneity and perinatal deathKenji Amano, Morimitsu Fujii, Satoru Arata, et al.Scientific Reports|August 27, 2020
Impairment of spatial memory accuracy improved by Cbr1 copy number resumption and GABAB receptor-dependent enhancement of synaptic inhibition in Down syndrome model miceFumiko Arima-Yoshida, Matthieu Raveau, Atsushi Shimohata, et al.Human Molecular Genetics|August 8, 2006
Mitochondrial dysfunction and tau hyperphosphorylation in Ts1Cje, a mouse model for Down syndromeEbrahim Abdul Shukkur, Atsushi Shimohata, Takumi Akagi, et al.Nature Communications|April 25, 2019
Impaired cortico-striatal excitatory transmission triggers epilepsyHiroyuki Miyamoto, Tetsuya Tatsukawa, Atsushi Shimohata, et al.Experimental Neurology|March 25, 2017
Ts1Cje Down syndrome model mice exhibit environmental stimuli-triggered locomotor hyperactivity and sociability concurrent with increased flux through central dopamine and serotonin metabolismAtsushi Shimohata, Keiichi Ishihara, Satoko Hattori, et al.Scientific Reports|May 17, 2022
CUX2 deficiency causes facilitation of excitatory synaptic transmission onto hippocampus and increased seizure susceptibility to kainateToshimitsu Suzuki, Tetsuya Tatsukawa, Genki Sudo, et al.Annals of Clinical and Translational Neurology|June 13, 2020
A recurrent PJA1 variant in trigonocephaly and neurodevelopmental disordersToshimitsu Suzuki, Toshifumi Suzuki, Matthieu Raveau, et al.Pageof 2