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Audrey M G Ragagnin

Showing results (1-10 of 9) with videos related to

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Frontiers in Neuroscience|July 19, 2019
Motor Neuron Susceptibility in ALS/FTDAudrey M G Ragagnin, Sina Shadfar, Marta Vidal, et al.
Cellular and Molecular Life Sciences : CMLS|December 6, 2019
Amyotrophic lateral sclerosis-linked UBQLN2 mutants inhibit endoplasmic reticulum to Golgi transport, leading to Golgi fragmentation and ER stressMark Halloran, Audrey M G Ragagnin, Marta Vidal, et al.
Human Molecular Genetics|February 7, 2018
ERp57 is protective against mutant SOD1-induced cellular pathology in amyotrophic lateral sclerosisSonam Parakh, Cyril J Jagaraj, Marta Vidal, et al.
Molecular Neurobiology|May 9, 2024
C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTDJessica Sultana, Audrey M G Ragagnin, Sonam Parakh, et al.
Iscience|May 24, 2020
The Redox Activity of Protein Disulfide Isomerase Inhibits ALS Phenotypes in Cellular and Zebrafish ModelsSonam Parakh, Sina Shadfar, Emma R Perri, et al.
Brain : a Journal of Neurology|March 10, 2026
Cofilin hyperphosphorylation triggers TDP-43 pathology in sporadic amyotrophic lateral sclerosisCyril Jones Jagaraj, Sayanthooran Saravanabavan, Sonam Parakh, et al.
Scientific Reports|November 22, 2023
ALS/FTD-associated mutation in cyclin F inhibits ER-Golgi trafficking, inducing ER stress, ERAD and Golgi fragmentationAudrey M G Ragagnin, Vinod Sundaramoorthy, Fabiha Farzana, et al.
Molecular Neurodegeneration|September 10, 2020
Impaired NHEJ repair in amyotrophic lateral sclerosis is associated with TDP-43 mutationsAnna Konopka, Donna R Whelan, Md Shafi Jamali, et al.
Brain : a Journal of Neurology|March 19, 2020
CYLD is a causative gene for frontotemporal dementia - amyotrophic lateral sclerosisCarol Dobson-Stone, Marianne Hallupp, Hamideh Shahheydari, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
Frontiers in Neuroscience|July 19, 2019
Motor Neuron Susceptibility in ALS/FTDAudrey M G Ragagnin, Sina Shadfar, Marta Vidal, et al.
Cellular and Molecular Life Sciences : CMLS|December 6, 2019
Amyotrophic lateral sclerosis-linked UBQLN2 mutants inhibit endoplasmic reticulum to Golgi transport, leading to Golgi fragmentation and ER stressMark Halloran, Audrey M G Ragagnin, Marta Vidal, et al.
Human Molecular Genetics|February 7, 2018
ERp57 is protective against mutant SOD1-induced cellular pathology in amyotrophic lateral sclerosisSonam Parakh, Cyril J Jagaraj, Marta Vidal, et al.
Molecular Neurobiology|May 9, 2024
C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTDJessica Sultana, Audrey M G Ragagnin, Sonam Parakh, et al.
Iscience|May 24, 2020
The Redox Activity of Protein Disulfide Isomerase Inhibits ALS Phenotypes in Cellular and Zebrafish ModelsSonam Parakh, Sina Shadfar, Emma R Perri, et al.
Brain : a Journal of Neurology|March 10, 2026
Cofilin hyperphosphorylation triggers TDP-43 pathology in sporadic amyotrophic lateral sclerosisCyril Jones Jagaraj, Sayanthooran Saravanabavan, Sonam Parakh, et al.
Scientific Reports|November 22, 2023
ALS/FTD-associated mutation in cyclin F inhibits ER-Golgi trafficking, inducing ER stress, ERAD and Golgi fragmentationAudrey M G Ragagnin, Vinod Sundaramoorthy, Fabiha Farzana, et al.
Molecular Neurodegeneration|September 10, 2020
Impaired NHEJ repair in amyotrophic lateral sclerosis is associated with TDP-43 mutationsAnna Konopka, Donna R Whelan, Md Shafi Jamali, et al.
Brain : a Journal of Neurology|March 19, 2020
CYLD is a causative gene for frontotemporal dementia - amyotrophic lateral sclerosisCarol Dobson-Stone, Marianne Hallupp, Hamideh Shahheydari, et al.
Pageof 1