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Proceedings of the National Academy of Sciences of the United States of America|June 12, 2012
Anoctamin 1 (Tmem16A) Ca2+-activated chloride channel stoichiometrically interacts with an ezrin-radixin-moesin networkPatricia Perez-Cornejo, Avanti Gokhale, Charity Duran, et al.Iscience|May 21, 2020
Cdh1-APC Regulates Protein Synthesis and Stress Granules in Neurons through an FMRP-Dependent MechanismArielle N Valdez-Sinon, Austin Lai, Liang Shi, et al.The Journal of Biological Chemistry|April 10, 2014
Mutations in the BLOC-1 subunits dysbindin and muted generate divergent and dosage-dependent phenotypesJennifer Larimore, Stephanie A Zlatic, Avanti Gokhale, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 17, 2012
Quantitative proteomic and genetic analyses of the schizophrenia susceptibility factor dysbindin identify novel roles of the biogenesis of lysosome-related organelles complex 1Avanti Gokhale, Jennifer Larimore, Erica Werner, et al.Human Molecular Genetics|July 23, 2015
Neuronal copper homeostasis susceptibility by genetic defects in dysbindin, a schizophrenia susceptibility factorAvanti Gokhale, Alysia Vrailas-Mortimer, Jennifer Larimore, et al.Molecular Biology of the Cell|October 15, 2011
The schizophrenia susceptibility factor dysbindin and its associated complex sort cargoes from cell bodies to the synapseJennifer Larimore, Karine Tornieri, Pearl V Ryder, et al.Eneuro|July 27, 2021
Heterogeneous Expression of Nuclear Encoded Mitochondrial Genes Distinguishes Inhibitory and Excitatory NeuronsMeghan E Wynne, Alicia R Lane, Kaela S Singleton, et al.Current Biology : CB|January 4, 2016
BLOC-1 Brings Together the Actin and Microtubule Cytoskeletons to Generate Recycling EndosomesCédric Delevoye, Xavier Heiligenstein, Léa Ripoll, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 15, 2015
The N-ethylmaleimide-sensitive factor and dysbindin interact to modulate synaptic plasticityAvanti Gokhale, Ariana P Mullin, Stephanie A Zlatic, et al.Biorxiv : the Preprint Server for Biology|February 6, 2026
Distinct signaling mechanisms and proteome phenotypes are elicited by compartment-specific genetic defects of copper homeostasisAlicia R Lane, Nadia Gonzalez, Avanti Gokhale, et al.Pageof 4