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American Journal of Physiology. Lung Cellular and Molecular Physiology|April 18, 2025
Oxidation of low-density lipoprotein by hemoglobin causes pulmonary microvascular endothelial barrier dysfunction through lectin-like oxidized LDL receptor 1Jamie E Meegan, Kyle J Riedmann, Samantha Gonski, et al.Scientific Reports|January 20, 2018
Cytokine-mediated changes in K+ channel activity promotes an adaptive Ca2+ response that sustains β-cell insulin secretion during inflammationMatthew T Dickerson, Avery M Bogart, Molly K Altman, et al.Microcirculation (New York, N.Y. : 1994)|May 21, 2025
Oxidized Cell-Free Hemoglobin Induces Mitochondrial Dysfunction by Activation of the Mitochondrial Permeability Transition Pore in the Pulmonary MicrovasculatureKyle J Riedmann, Jamie E Meegan, Aqeela Afzal, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|December 22, 2021
A two-hit model of sepsis plus hyperoxia causes lung permeability and inflammationJulie A Bastarache, Kyle Smith, Jordan J Jesse, et al.Endocrinology|September 22, 2018
Reducing Glucokinase Activity Restores Endogenous Pulsatility and Enhances Insulin Secretion in Islets From db/db MiceIshrat Jahan, Kathryn L Corbin, Avery M Bogart, et al.CHEST Critical Care|October 18, 2024
Elevated Hemoglobin A1c and the Risk of Developing ARDS in Two Cohort StudiesAvery M Bogart, Christine R Lopez, Sarah N Obeidalla, et al.Pageof 1