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Nature Communications|April 5, 2022
Opposite physiological and pathological mTORC1-mediated roles of the CB1 receptor in regulating renal tubular functionLiad Hinden, Majdoleen Ahmad, Sharleen Hamad, et al.
Molecular Metabolism|June 23, 2025
Metabolic consequences of altered kidney glucose reabsorption under normoglycemic conditionsMajdoleen Ahmad, Anna Permyakova, Saja Baraghithy, et al.
The Journal of Biological Chemistry|September 11, 2013
Protection of a ceramide synthase 2 null mouse from drug-induced liver injury: role of gap junction dysfunction and connexin 32 mislocalizationWoo-Jae Park, Joo-Won Park, Racheli Erez-Roman, et al.
Journal of the American Society of Nephrology : JASN|January 11, 2024
Renal Mitochondrial ATP Transporter Ablation Ameliorates Obesity-Induced CKDAnna Permyakova, Sharleen Hamad, Liad Hinden, et al.
JCI Insight|February 22, 2023
Mapping the metabolic reprogramming induced by sodium-glucose cotransporter 2 inhibitionAviram Kogot-Levin, Yael Riahi, Ifat Abramovich, et al.
The Journal of Biological Chemistry|June 28, 2011
Encephalopathy caused by ablation of very long acyl chain ceramide synthesis may be largely due to reduced galactosylceramide levelsOshrit Ben-David, Yael Pewzner-Jung, Ori Brenner, et al.
JCI Insight|May 22, 2026
Glucosamine links hyperglycemia to mTORC1 activation and glucose toxicity in diabetesYael Riahi, Aviram Kogot-Levin, Ziv Teselpapa, et al.
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