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International Journal of Molecular Sciences|June 22, 2018
Yeast-Based Screen to Identify Natural Compounds with a Potential Therapeutic Effect in Hailey-Hailey DiseaseGraziella Ficociello, Azzurra Zonfrilli, Samantha Cialfi, et al.
International Journal of Molecular Sciences|March 6, 2019
DNA Damage Stress: Cui Prodest?Nagendra Verma, Matteo Franchitto, Azzurra Zonfrilli, et al.
Molecules (Basel, Switzerland)|December 11, 2019
Hypotonic, Acidic Oxidizing Solution Containing Hypochlorous Acid (HClO) as a Potential Treatment of Hailey-Hailey DiseaseSamantha Cialfi, Salvatore Calabro, Matteo Franchitto, et al.
Scientific Reports|August 17, 2016
The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey diseaseSamantha Cialfi, Loredana Le Pera, Carlo De Blasio, et al.
Cell Death Discovery|April 13, 2021
Functional cooperation between ASK1 and p21<sup>Waf1/Cip1</sup> in the balance of cell-cycle arrest, cell death and tumorigenesis of stressed keratinocytesCarlo De Blasio, Nagendra Verma, Marta Moretti, et al.
The Journal of Biological Chemistry|October 11, 2019
PLK1 targets NOTCH1 during DNA damage and mitotic progressionCarlo De Blasio, Azzurra Zonfrilli, Matteo Franchitto, et al.
Experimental Dermatology|February 15, 2023
Loss of ATP2C1 function promotes trafficking and degradation of NOTCH1: Implications for Hailey-Hailey diseaseAzzurra Zonfrilli, Federica Truglio, Alessandra Simeone, et al.
Nature Communications|July 2, 2025
PI3Kγ signaling controls trafficking of CD8<sup>+</sup> T cells between lymphoid and non-lymphoid organs and drives hypertension in a murine modelMarialuisa Perrotta, Sara Perrotta, Lorenzo Carnevale, et al.
Immunity|March 1, 2025
A heart-brain-spleen axis controls cardiac remodeling to hypertensive stressSara Perrotta, Lorenzo Carnevale, Marialuisa Perrotta, et al.
Cardiovascular Research|April 9, 2025
Beta 2 adrenergic receptor gene methylation activates innate lymphoid cells to drive hypertension in lymphocyte deficient hostsWei Chen, Sara Perrotta, Liang Xiao, et al.
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