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The Journal of Comparative Neurology
|
July 22, 1998
Amyloid beta protein is internalized selectively by hippocampal field CA1 and causes neurons to accumulate amyloidogenic carboxyterminal fragments of the amyloid precursor protein
B A Bahr, K B Hoffman, A J Yang, et al.
Kidney International
|
November 14, 1997
Effect of glycine on prelethal and postlethal increases in calpain activity in rat renal proximal tubules
C L Edelstein, H Ling, P E Gengaro, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
February 15, 1997
Arg-Gly-Asp-Ser-selective adhesion and the stabilization of long-term potentiation: pharmacological studies and the characterization of a candidate matrix receptor
B A Bahr, U Staubli, P Xiao, et al.
Cell Death and Differentiation
|
December 14, 2004
The influence of age on apoptotic and other mechanisms of cell death after cerebral hypoxia-ischemia
C Zhu, X Wang, F Xu, et al.
The Journal of Biological Chemistry
|
January 2, 2001
Synergistic activation of caspase-3 by m-calpain after neonatal hypoxia-ischemia: a mechanism of "pathological apoptosis"?
K Blomgren, C Zhu, X Wang, et al.
Annals of the New York Academy of Sciences
|
January 1, 1987
Acetylcholine transport: fundamental properties and effects of pharmacologic agents
S M Parsons, B A Bahr, L M Gracz, et al.
Neuroscience
|
February 7, 2013
Spatiotemporal resolution of BDNF neuroprotection against glutamate excitotoxicity in cultured hippocampal neurons
C V Melo, S Okumoto, J R Gomes, et al.
The American Journal of Pathology
|
January 6, 2001
Correlation between caspase activation and neurofibrillary tangle formation in Alzheimer's disease
T T Rohn, E Head, J H Su, et al.
Hippocampus
|
January 1, 1995
Stable maintenance of glutamate receptors and other synaptic components in long-term hippocampal slices
B A Bahr, M Kessler, S Rivera, et al.
The Journal of Biological Chemistry
|
May 13, 1999
Calpastatin is up-regulated in response to hypoxia and is a suicide substrate to calpain after neonatal cerebral hypoxia-ischemia
K Blomgren, U Hallin, A L Andersson, et al.
Page
of 6
Search research articles
Search
Showing results (41-50 of 54) with videos related to
Sort By:
Page
of 6
The Journal of Comparative Neurology
|
July 22, 1998
Amyloid beta protein is internalized selectively by hippocampal field CA1 and causes neurons to accumulate amyloidogenic carboxyterminal fragments of the amyloid precursor protein
B A Bahr, K B Hoffman, A J Yang, et al.
Kidney International
|
November 14, 1997
Effect of glycine on prelethal and postlethal increases in calpain activity in rat renal proximal tubules
C L Edelstein, H Ling, P E Gengaro, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience
|
February 15, 1997
Arg-Gly-Asp-Ser-selective adhesion and the stabilization of long-term potentiation: pharmacological studies and the characterization of a candidate matrix receptor
B A Bahr, U Staubli, P Xiao, et al.
Cell Death and Differentiation
|
December 14, 2004
The influence of age on apoptotic and other mechanisms of cell death after cerebral hypoxia-ischemia
C Zhu, X Wang, F Xu, et al.
The Journal of Biological Chemistry
|
January 2, 2001
Synergistic activation of caspase-3 by m-calpain after neonatal hypoxia-ischemia: a mechanism of "pathological apoptosis"?
K Blomgren, C Zhu, X Wang, et al.
Annals of the New York Academy of Sciences
|
January 1, 1987
Acetylcholine transport: fundamental properties and effects of pharmacologic agents
S M Parsons, B A Bahr, L M Gracz, et al.
Neuroscience
|
February 7, 2013
Spatiotemporal resolution of BDNF neuroprotection against glutamate excitotoxicity in cultured hippocampal neurons
C V Melo, S Okumoto, J R Gomes, et al.
The American Journal of Pathology
|
January 6, 2001
Correlation between caspase activation and neurofibrillary tangle formation in Alzheimer's disease
T T Rohn, E Head, J H Su, et al.
Hippocampus
|
January 1, 1995
Stable maintenance of glutamate receptors and other synaptic components in long-term hippocampal slices
B A Bahr, M Kessler, S Rivera, et al.
The Journal of Biological Chemistry
|
May 13, 1999
Calpastatin is up-regulated in response to hypoxia and is a suicide substrate to calpain after neonatal cerebral hypoxia-ischemia
K Blomgren, U Hallin, A L Andersson, et al.
Page
of 6