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The Journal of Surgical Research|April 26, 2001
Clostridium difficile toxins and enterococcal translocation in vivo and in vitroB A Feltis, R M Garni, C L WellsClinical Transplantation|June 27, 1998
Acute acalculous cholecystitis (AAC) resulting in gallbladder perforation in a solid organ transplant recipient: a case reportB A Feltis, D A Lee, R W GruessnerShock (Augusta, Ga.)|July 1, 1994
Bacterial translocation and lipopolysaccharide-induced mortality in genetically macrophage-deficient op/op miceB A Feltis, R P Jechorek, S L Erlandsen, et al.Journal of Medical Microbiology|May 1, 1993
Oral infectivity and bacterial interactions with mononuclear phagocytesC L Wells, B A Feltis, D F Hanson, et al.Gastroenterology|May 1, 1996
Bacteroides fragilis enterotoxin modulates epithelial permeability and bacterial internalization by HT-29 enterocytesC L Wells, E M van de Westerlo, R P Jechorek, et al.Archives of Surgery (Chicago, Ill. : 1960)|November 11, 1999
Clostridium difficile toxins may augment bacterial penetration of intestinal epitheliumB A Feltis, A S Kim, K M Kinneberg, et al.Shock (Augusta, Ga.)|December 29, 2000
Clostridium difficile toxins A and B can alter epithelial permeability and promote bacterial paracellular migration through HT-29 enterocytesB A Feltis, S M Wiesner, A S Kim, et al.The Journal of Surgical Research|October 18, 2002
Liver and circulating NK1.1(+)CD3(-) cells are increased in infection with attenuated Salmonella typhimurium and are associated with reduced tumor in murine liver cancerB A Feltis, J S Miller, D A Sahar, et al.Pageof 1