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The Journal of Biological Chemistry|September 28, 2006
Proteolytic processing of OPA1 links mitochondrial dysfunction to alterations in mitochondrial morphologyStéphane Duvezin-Caubet, Ravi Jagasia, Johannes Wagener, et al.Life Science Alliance|October 11, 2024
Mitochondrial apolipoprotein MIC26 is a metabolic rheostat regulating central cellular fuel pathwaysMelissa Damiecki, Ritam Naha, Yulia Schaumkessel, et al.European Journal of Cancer (Oxford, England : 1990)|July 27, 2002
Phase I and pharmacological study of daily oral administration of perifosine (D-21266) in patients with advanced solid tumoursM Crul, H Rosing, G J de Klerk, et al.The EMBO Journal|October 15, 2019
Individual cristae within the same mitochondrion display different membrane potentials and are functionally independentDane M Wolf, Mayuko Segawa, Arun Kumar Kondadi, et al.Disease Models & Mechanisms|September 12, 2020
Ammonia inhibits energy metabolism in astrocytes in a rapid and glutamate dehydrogenase 2-dependent mannerLeonie Drews, Marcel Zimmermann, Philipp Westhoff, et al.Life Science Alliance|August 14, 2020
MIC26 and MIC27 cooperate to regulate cardiolipin levels and the landscape of OXPHOS complexesRuchika Anand, Arun Kumar Kondadi, Jana Meisterknecht, et al.EMBO Reports|December 17, 2009
Nix is a selective autophagy receptor for mitochondrial clearanceIvana Novak, Vladimir Kirkin, David G McEwan, et al.Blood|November 26, 1999
Interferon-alpha before allogeneic bone marrow transplantation in chronic myelogenous leukemia does not affect outcome adversely, provided it is discontinued at least 90 days before the procedureR Hehlmann, A Hochhaus, H J Kolb, et al.Bone Marrow Transplantation|May 1, 1996
Randomized study of the combination of hydroxyurea and interferon alpha versus hydroxyurea monotherapy during the chronic phase of chronic myelogenous leukemia (CML Study II). The German CML Study GroupR Hehlmann, H Heimpel, D K Hossfeld, et al.Stem Cell Research & Therapy|June 26, 2017
Modulation of oxidative phosphorylation and redox homeostasis in mitochondrial NDUFS4 deficiency via mesenchymal stem cellsMarlen Melcher, Katharina Danhauser, Annette Seibt, et al.Pageof 56