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Journal of Molecular and Cellular Cardiology|April 13, 1999
Reduced Ca(2+)-sensitivity of SERCA 2a in failing human myocardium due to reduced serin-16 phospholamban phosphorylationR H Schwinger, G Münch, B Bölck, et al.Journal of Molecular Medicine (Berlin, Germany)|June 13, 1998
Unchanged protein expression of sarcoplasmic reticulum Ca2+-ATPase, phospholamban, and calsequestrin in terminally failing human myocardiumG Münch, B Bölck, S Hoischen, et al.American Journal of Physiology. Heart and Circulatory Physiology|June 14, 2000
SERCA2a activity correlates with the force-frequency relationship in human myocardiumG Münch, B Bölck, K Brixius, et al.The Journal of Pharmacology and Experimental Therapeutics|October 23, 1997
Effect of cyclopiazonic acid on the force-frequency relationship in human nonfailing myocardiumR H Schwinger, K Brixius, U Bavendiek, et al.Molecular and Cellular Biochemistry|May 14, 2008
Reduced troponin I phosphorylation and increased Ca(2+)-dependent ATP-consumption in triton X-skinned fiber preparations from Galphaq overexpressor miceC Pott, L Willkomm, S Grafweg, et al.British Journal of Pharmacology|February 6, 2003
The preferential beta3-adrenoceptor agonist BRL 37344 increases force via beta1-/beta2-adrenoceptors and induces endothelial nitric oxide synthase via beta3-adrenoceptors in human atrial myocardiumC Pott, K Brixius, A Bundkirchen, et al.Basic Research in Cardiology|April 7, 2000
The enhanced contractility in phospholamban deficient mouse hearts is not associated with alterations in (Ca2+)-sensitivity or myosin ATPase-activity of the contractile proteinsR H Schwinger, K Brixius, P Savvidou-Zaroti, et al.Canadian Journal of Physiology and Pharmacology|March 19, 2010
eNOS phosphorylation and translocation are altered in male but not female mice by increased activation of the Gαq proteinC Ruiz-Holst, B Bölck, A Ghanem, et al.Journal of Molecular and Cellular Cardiology|April 6, 2005
Overexpression of sorcin enhances cardiac contractility in vivo and in vitroK F Frank, B Bölck, Z Ding, et al.Pageof 2