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The Journal of Clinical Endocrinology and Metabolism
|
October 16, 1999
Diagnostic value of fluorometric assays in the evaluation of precocious puberty
V N Brito, M C Batista, M F Borges, et al.
Human Heredity
|
April 3, 1999
Low frequency of CYP2B deletions in Brazilian patients with congenital adrenal hyperplasia due to 21-hydroxylas deficiency
T A Bachega, A E Billerbeck, G Madureira, et al.
Nature Genetics
|
May 1, 1994
Male pseudohermaphroditism caused by mutations of testicular 17 beta-hydroxysteroid dehydrogenase 3
W M Geissler, D L Davis, L Wu, et al.
The Journal of Clinical Endocrinology and Metabolism
|
January 3, 2001
Gonadotropin-independent precocious puberty due to luteinizing hormone receptor mutations in Brazilian boys: a novel constitutively activating mutation in the first transmembrane helix
A C Latronico, H Shinozaki, G Guerra, et al.
The Journal of Clinical Endocrinology and Metabolism
|
August 12, 1999
A novel missense mutation, GLY424SER, in Brazilian patients with 21-hydroxylase deficiency
A E Billerbeck, T A Bachega, E T Frazatto, et al.
Clinical Genetics
|
November 26, 2009
Molecular analysis of CYP21A2 can optimize the follow-up of positive results in newborn screening for congenital adrenal hyperplasia
E L Silveira, R H Elnecave, E P dos Santos, et al.
Journal of Hypertension
|
February 12, 1998
Apparent mineralocorticoid excess in a Brazilian kindred: hypertension in the heterozygote state
A Li, K X Li, S Marui, et al.
Medicine
|
October 20, 2000
Male pseudohermaphroditism due to 17 beta-hydroxysteroid dehydrogenase 3 deficiency. Diagnosis, psychological evaluation, and management
B B Mendonca, M Inacio, I J Arnhold, et al.
Clinical Endocrinology
|
August 22, 2006
Combined pituitary hormone deficiency (CPHD) due to a complete PROP1 deletion
M G Abrão, M V Leite, L R Carvalho, et al.
The Journal of Clinical Endocrinology and Metabolism
|
June 17, 1998
Activating mutation of the stimulatory G protein (gsp) as a putative cause of ovarian and testicular human stromal Leydig cell tumors
M C Fragoso, A C Latronico, F M Carvalho, et al.
Page
of 9
Search research articles
Search
Showing results (61-70 of 82) with videos related to
Sort By:
Page
of 9
The Journal of Clinical Endocrinology and Metabolism
|
October 16, 1999
Diagnostic value of fluorometric assays in the evaluation of precocious puberty
V N Brito, M C Batista, M F Borges, et al.
Human Heredity
|
April 3, 1999
Low frequency of CYP2B deletions in Brazilian patients with congenital adrenal hyperplasia due to 21-hydroxylas deficiency
T A Bachega, A E Billerbeck, G Madureira, et al.
Nature Genetics
|
May 1, 1994
Male pseudohermaphroditism caused by mutations of testicular 17 beta-hydroxysteroid dehydrogenase 3
W M Geissler, D L Davis, L Wu, et al.
The Journal of Clinical Endocrinology and Metabolism
|
January 3, 2001
Gonadotropin-independent precocious puberty due to luteinizing hormone receptor mutations in Brazilian boys: a novel constitutively activating mutation in the first transmembrane helix
A C Latronico, H Shinozaki, G Guerra, et al.
The Journal of Clinical Endocrinology and Metabolism
|
August 12, 1999
A novel missense mutation, GLY424SER, in Brazilian patients with 21-hydroxylase deficiency
A E Billerbeck, T A Bachega, E T Frazatto, et al.
Clinical Genetics
|
November 26, 2009
Molecular analysis of CYP21A2 can optimize the follow-up of positive results in newborn screening for congenital adrenal hyperplasia
E L Silveira, R H Elnecave, E P dos Santos, et al.
Journal of Hypertension
|
February 12, 1998
Apparent mineralocorticoid excess in a Brazilian kindred: hypertension in the heterozygote state
A Li, K X Li, S Marui, et al.
Medicine
|
October 20, 2000
Male pseudohermaphroditism due to 17 beta-hydroxysteroid dehydrogenase 3 deficiency. Diagnosis, psychological evaluation, and management
B B Mendonca, M Inacio, I J Arnhold, et al.
Clinical Endocrinology
|
August 22, 2006
Combined pituitary hormone deficiency (CPHD) due to a complete PROP1 deletion
M G Abrão, M V Leite, L R Carvalho, et al.
The Journal of Clinical Endocrinology and Metabolism
|
June 17, 1998
Activating mutation of the stimulatory G protein (gsp) as a putative cause of ovarian and testicular human stromal Leydig cell tumors
M C Fragoso, A C Latronico, F M Carvalho, et al.
Page
of 9