Showing results (11-20 of 1,817) with videos related to
Sort By:
Pageof 182
Clinical and Applied Thrombosis/Hemostasis : Official Journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis|February 24, 2001
A comparison of every-third-day versus daily low-dose aspirin therapy on serum thromboxane concentrations in healthy men and womenM Feldman, B Cryer, K Rushin, et al.Journal of the American College of Cardiology|June 23, 2001
Effects of low-dose aspirin on serum C-reactive protein and thromboxane B2 concentrations: a placebo-controlled study using a highly sensitive C-reactive protein assayM Feldman, I Jialal, S Devaraj, et al.The American Journal of Gastroenterology|June 23, 2001
Role of Helicobacter pylori infection in gastroduodenal injury and gastric prostaglandin synthesis during long term/low dose aspirin therapy: a prospective placebo-controlled, double-blind randomized trialM Feldman, B Cryer, D Mallat, et al.JAMA|August 1, 1998
Role of seroconversion in confirming cure of Helicobacter pylori infectionM Feldman, B Cryer, E Lee, et al.Gastroenterology|December 1, 1990
Comparison of salsalate and aspirin on mucosal injury and gastroduodenal mucosal prostaglandinsB Cryer, M Goldschmiedt, J S Redfern, et al.Proceedings of the Association of American Physicians|October 16, 1999
Effects of cutaneous aspirin on the human stomach and duodenumB Cryer, D Kliewer, H Sie, et al.Gastroenterology|April 1, 1992
Effect of aging on gastric and duodenal mucosal prostaglandin concentrations in humansB Cryer, J S Redfern, M Goldschmiedt, et al.The American Journal of Physiology|December 22, 1999
Influence of H. pylori infection on meal-stimulated gastric acid secretion and gastroesophageal acid refluxM Feldman, B Cryer, D Sammer, et al.Gastroenterology|April 1, 1996
Effects of aging and gastritis on gastric acid and pepsin secretion in humans: a prospective studyM Feldman, B Cryer, K E McArthur, et al.The American Journal of Gastroenterology|December 1, 1992
Gastric and duodenal mucosal prostaglandin concentrations in gastric or duodenal ulcer disease: relationships with demographics, environmental, and histological factors, including Helicobacter pyloriB Cryer, T W Faust, M Goldschmiedt, et al.Pageof 182