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Molecular Medicine (Cambridge, Mass.)|September 29, 1999

Developmental expression of wild-type and mutant presenilin-1 in hippocampal neurons from transgenic mice: evidence for novel species-specific properties of human presenilin-1L Lévesque, W Annaert, K Craessaerts, et al.

The EMBO Journal|October 1, 1996

The beta-amyloid domain is essential for axonal sorting of amyloid precursor proteinP J Tienari, B De Strooper, E Ikonen, et al.

Nature|October 28, 1998

Destabilization of beta-catenin by mutations in presenilin-1 potentiates neuronal apoptosisZ Zhang, H Hartmann, V M Do, et al.

Human Molecular Genetics|November 7, 2000

Nonfibrillar diffuse amyloid deposition due to a gamma(42)-secretase site mutation points to an essential role for N-truncated A beta(42) in Alzheimer's diseaseS Kumar-Singh, C De Jonghe, M Cruts, et al.

Human Mutation|August 19, 2006

Alzheimer dementia caused by a novel mutation located in the APP C-terminal intracytosolic fragmentJ Theuns, E Marjaux, M Vandenbulcke, et al.

Proceedings of the National Academy of Sciences of the United States of America|October 16, 1999

Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiencyA Herreman, D Hartmann, W Annaert, et al.

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Showing results (61-70 of 66) with videos related to

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You have reached the last page of results.This site can display upto 66 results.

Molecular Medicine (Cambridge, Mass.)|September 29, 1999

Developmental expression of wild-type and mutant presenilin-1 in hippocampal neurons from transgenic mice: evidence for novel species-specific properties of human presenilin-1L Lévesque, W Annaert, K Craessaerts, et al.

The EMBO Journal|October 1, 1996

The beta-amyloid domain is essential for axonal sorting of amyloid precursor proteinP J Tienari, B De Strooper, E Ikonen, et al.

Nature|October 28, 1998

Destabilization of beta-catenin by mutations in presenilin-1 potentiates neuronal apoptosisZ Zhang, H Hartmann, V M Do, et al.

Human Molecular Genetics|November 7, 2000

Nonfibrillar diffuse amyloid deposition due to a gamma(42)-secretase site mutation points to an essential role for N-truncated A beta(42) in Alzheimer's diseaseS Kumar-Singh, C De Jonghe, M Cruts, et al.

Human Mutation|August 19, 2006

Alzheimer dementia caused by a novel mutation located in the APP C-terminal intracytosolic fragmentJ Theuns, E Marjaux, M Vandenbulcke, et al.

Proceedings of the National Academy of Sciences of the United States of America|October 16, 1999

Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiencyA Herreman, D Hartmann, W Annaert, et al.

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