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The Journal of Clinical Investigation|April 1, 1990
Release of Ca2+ from the endoplasmic reticulum is not the mechanism for bile acid-induced cholestasis and hepatotoxicity in the intact rat liverG C Farrell, S K Duddy, G E Kass, et al.Biochemical Pharmacology|October 15, 1985
On the role of thiol groups in the inhibition of liver microsomal Ca2+ sequestration by toxic agentsH Thor, P Hartzell, S A Svensson, et al.European Journal of Biochemistry|April 15, 1995
Adenosine inhibits protein synthesis in isolated rat hepatocytes. Evidence for a lack of involvement of intracellular calcium in the mechanism of inhibitionS A Tinton, S C Chow, P M Buc-Calderon, et al.Cell Death and Differentiation|May 1, 1997
Inorganic mercury modifies Ca2+ signals, triggers apoptosis and potentiates NMDA toxicity in cerebellar granule neuronsA D Rossi, B Viviani, B Zhivotovsky, et al.Biochemical Pharmacology|June 9, 1993
Eicosanoids released following inhibition of the endoplasmic reticulum Ca2+ pump stimulate Ca2+ efflux in the perfused rat liverJ Llopis, G C Farrell, S K Duddy, et al.FEBS Letters|May 8, 1995
Involvement of multiple proteases during Fas-mediated apoptosis in T lymphocytesS C Chow, M Weis, G E Kass, et al.Chemical Research in Toxicology|June 1, 1997
Mechanism of dithiocarbamate inhibition of apoptosis: thiol oxidation by dithiocarbamate disulfides directly inhibits processing of the caspase-3 proenzymeC S Nobel, D H Burgess, B Zhivotovsky, et al.Free Radical Research Communications|January 1, 1990
2,5-Di(tert-butyl)-1,4-benzohydroquinone--a novel mobilizer of the inositol 1,4,5-trisphosphate-sensitive Ca2+ poolG A Moore, G E Kass, S K Duddy, et al.Toxicology and Applied Pharmacology|June 7, 2008
Increased accumulation of neutrophils and decreased fibrosis in the lung of NADPH oxidase-deficient C57BL/6 mice exposed to carbon nanotubesA A Shvedova, E R Kisin, A R Murray, et al.Cell Death and Differentiation|April 14, 1999
Human skeletal muscle cytosols are refractory to cytochrome c-dependent activation of type-II caspases and lack APAF-1D H Burgess, M Svensson, T Dandrea, et al.Pageof 30