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Tissue Antigens|March 1, 1991
Distribution of HLA-DRB1, -DQA1 and -DQB1 alleles and DQA1-DQB1 genotypes among Norwegian patients with insulin-dependent diabetes mellitusK S Rønningen, A Spurkland, T Iwe, et al.
The Journal of Experimental Medicine|November 1, 1977
The genetic polymorphism of the fourth component of human complement: methodological aspects and a presentation of linkage and association data relevant to its localization in the HLA regionP Teisberg, B Olaisen, R Jonassen, et al.
Bailliere'S Clinical Endocrinology and Metabolism|September 1, 1991
Insulin dependent diabetes mellitus susceptibility or protection may be determined by certain HLA-DQ moleculesE Thorsby, H A Gjertsen, K E Lundin, et al.
Human Immunology|November 1, 1992
HLA-DR and -DQ genotypes of celiac disease patients serologically typed to be non-DR3 or non-DR5/7A Spurkland, L M Sollid, I Polanco, et al.
Annals of Human Genetics|October 1, 1983
Gene order and gene distances in the HLA region studied by the haplotype methodB Olaisen, P Teisberg, R Jonassen, et al.
Human Immunology|June 1, 1989
Patients with multiple sclerosis carry DQB1 genes which encode shared polymorphic amino acid sequencesF Vartdal, L M Sollid, B Vandvik, et al.
Human Immunology|January 1, 1991
HLA-DQA1 and HLA-DQB1 genes may jointly determine susceptibility to develop multiple sclerosisA Spurkland, K S Rønningen, B Vandvik, et al.
Transfusion|March 3, 1998
Effect of filtration and storage of platelet concentrates on the production of the chemotaxins C5a, interleukin 8, tumor necrosis factor alpha, and leukotriene B4G Hetland, T E Mollnes, K Bergh, et al.
Transfusion and Apheresis Science : Official Journal of the World Apheresis Association : Official Journal of the European Society for Haemapheresis|August 6, 2004
Transferrin receptor in serum. A new tool in the diagnosis and prevention of iron deficiency in blood donorsO Flesland, A-K Eskelund, A B Flesland, et al.
The Journal of Rheumatology|August 1, 1994
Seronegative and weakly seropositive rheumatoid arthritis differ from clearly seropositive rheumatoid arthritis in HLA class II associationsR Ploski, O J Mellbye, K S Rønningen, et al.
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