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AIDS (London, England)|December 20, 2003
Influence of HLA-B57 on clinical presentation and viral control during acute HIV-1 infectionMarcus Altfeld, Marylyn M Addo, Eric S Rosenberg, et al.Expert Review of Gastroenterology & Hepatology|March 10, 2017
Effectiveness of interferon-free therapy for the treatment of HCV-patients with compensated cirrhosis treated through the Irish early access programE Gray, A O'Leary, C Bergin, et al.Journal of Virology|January 15, 2003
Comprehensive epitope analysis of human immunodeficiency virus type 1 (HIV-1)-specific T-cell responses directed against the entire expressed HIV-1 genome demonstrate broadly directed responses, but no correlation to viral loadM M Addo, X G Yu, A Rathod, et al.Proceedings of the National Academy of Sciences of the United States of America|July 20, 2022
Genetic variation that determines TAPBP expression levels associates with the course of malaria in an HLA allotype-dependent mannerVictoria Walker-Sperling, Jean C Digitale, Mathias Viard, et al.Plos One|September 2, 2016
A Novel Model of Asymptomatic Plasmodium Parasitemia That Recapitulates Elements of the Human Immune Response to Chronic InfectionMary F Fontana, Alyssa Baccarella, Joshua F Craft, et al.Retrovirology|June 13, 2026
Metabolic reprogramming of CD4⁺ T cells by Zaprinast induces HIV-1 latency reversal ex vivoV De Masson d'Autume, V LeDouce, A Dutilleul, et al.The Journal of Experimental Medicine|April 7, 2004
Immune selection for altered antigen processing leads to cytotoxic T lymphocyte escape in chronic HIV-1 infectionRika Draenert, Sylvie Le Gall, Katja J Pfafferott, et al.Journal of Virology|July 30, 2005
HLA-B63 presents HLA-B57/B58-restricted cytotoxic T-lymphocyte epitopes and is associated with low human immunodeficiency virus loadNicole Frahm, Sharon Adams, Photini Kiepiela, et al.Plos Pathogens|July 17, 2015
Decline of FoxP3+ Regulatory CD4 T Cells in Peripheral Blood of Children Heavily Exposed to MalariaMichelle J Boyle, Prasanna Jagannathan, Lila A Farrington, et al.Journal of Virology|October 18, 2005
Selective escape from CD8+ T-cell responses represents a major driving force of human immunodeficiency virus type 1 (HIV-1) sequence diversity and reveals constraints on HIV-1 evolutionTodd M Allen, Marcus Altfeld, Shaun C Geer, et al.Pageof 20