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Circulation|February 27, 2001
Notched T waves on Holter recordings enhance detection of patients with LQt2 (HERG) mutationsJ M Lupoglazoff, I Denjoy, M Berthet, et al.
Diabetes/Metabolism Research and Reviews|June 11, 1999
Peroxisome proliferator activated receptor-gamma, leptin and tumor necrosis factor-alpha mRNA expression during very low calorie diet in subcutaneous adipose tissue in obese womenJ P Bastard, B Hainque, E Dusserre, et al.
European Heart Journal|October 29, 1998
Diagnostic value of electrocardiography and echocardiography for familial hypertrophic cardiomyopathy in genotyped childrenP Charron, O Dubourg, M Desnos, et al.
Circulation|March 23, 1999
C-terminal HERG mutations: the role of hypokalemia and a KCNQ1-associated mutation in cardiac event occurrenceM Berthet, I Denjoy, C Donger, et al.
Genetic Counseling (Geneva, Switzerland)|January 1, 1997
Penetrance of familial hypertrophic cardiomyopathyP Charron, L Carrier, O Dubourg, et al.
Nature Genetics|December 1, 1995
Cardiac myosin binding protein-C gene splice acceptor site mutation is associated with familial hypertrophic cardiomyopathyG Bonne, L Carrier, J Bercovici, et al.
Circulation|June 19, 1998
Clinical features and prognostic implications of familial hypertrophic cardiomyopathy related to the cardiac myosin-binding protein C geneP Charron, O Dubourg, M Desnos, et al.
European Journal of Clinical Investigation|April 12, 2000
Subcutaneous adipose tissue expression of tumour necrosis factor-alpha is not associated with whole body insulin resistance in obese nondiabetic or in type-2 diabetic subjectsH A Koistinen, J P Bastard, E Dusserre, et al.
Circulation|December 15, 1996
Codon 102 of the cardiac troponin T gene is a putative hot spot for mutations in familial hypertrophic cardiomyopathyJ F Forissier, L Carrier, H Farza, et al.
Human Mutation|November 26, 1998
Genotype-phenotype analysis in four families with mutations in beta-myosin heavy chain gene responsible for familial hypertrophic cardiomyopathyF Tesson, P Richard, P Charron, et al.
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