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Diabetes
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October 9, 2013
Podocyte-specific GLUT4-deficient mice have fewer and larger podocytes and are protected from diabetic nephropathy
Johanna Guzman, Alexandra N Jauregui, Sandra Merscher-Gomez, et al.
The Journal of Clinical Investigation
|
May 2, 2022
Loss of the collagen IV modifier prolyl 3-hydroxylase 2 causes thin basement membrane nephropathy
Hande Aypek, Christoph Krisp, Shun Lu, et al.
Kidney International Reports
|
February 25, 2026
Effect of Steroids on Anti-nephrin Autoantibodies and B cells in Minimal Change Disease
Felicitas E Hengel, Asma Beldi-Ferchiou, Hamza Sakhi, et al.
JCI Insight
|
July 19, 2016
A flexible, multilayered protein scaffold maintains the slit in between glomerular podocytes
Florian Grahammer, Christoph Wigge, Christoph Schell, et al.
Journal of Cachexia, Sarcopenia and Muscle
|
March 28, 2019
Compression of morbidity in a progeroid mouse model through the attenuation of myostatin/activin signalling
Khalid Alyodawi, Wilbert P Vermeij, Saleh Omairi, et al.
Plos Pathogens
|
April 21, 2022
Th17 cell plasticity towards a T-bet-dependent Th1 phenotype is required for bacterial control in Staphylococcus aureus infection
Patricia Bartsch, Christoph Kilian, Malte Hellmig, et al.
Acta Neuropathologica
|
January 20, 2024
Temporal change of DNA methylation subclasses between matched newly diagnosed and recurrent glioblastoma
Richard Drexler, Robin Khatri, Ulrich Schüller, et al.
Journal of Cachexia, Sarcopenia and Muscle
|
September 9, 2024
Improved health by combining dietary restriction and promoting muscle growth in DNA repair-deficient progeroid mice
Wilbert P Vermeij, Khalid Alyodawi, Ivar van Galen, et al.
The New England Journal of Medicine
|
May 14, 2020
Multiorgan and Renal Tropism of SARS-CoV-2
Victor G Puelles, Marc Lütgehetmann, Maja T Lindenmeyer, et al.
Kidney International
|
March 28, 2014
Renal fibrosis is the common feature of autosomal dominant tubulointerstitial kidney diseases caused by mutations in mucin 1 or uromodulin
Arif B Ekici, Thomas Hackenbeck, Vincent Morinière, et al.
Page
of 64
Search research articles
Search
Showing results (521-530 of 633) with videos related to
Sort By:
Page
of 64
Diabetes
|
October 9, 2013
Podocyte-specific GLUT4-deficient mice have fewer and larger podocytes and are protected from diabetic nephropathy
Johanna Guzman, Alexandra N Jauregui, Sandra Merscher-Gomez, et al.
The Journal of Clinical Investigation
|
May 2, 2022
Loss of the collagen IV modifier prolyl 3-hydroxylase 2 causes thin basement membrane nephropathy
Hande Aypek, Christoph Krisp, Shun Lu, et al.
Kidney International Reports
|
February 25, 2026
Effect of Steroids on Anti-nephrin Autoantibodies and B cells in Minimal Change Disease
Felicitas E Hengel, Asma Beldi-Ferchiou, Hamza Sakhi, et al.
JCI Insight
|
July 19, 2016
A flexible, multilayered protein scaffold maintains the slit in between glomerular podocytes
Florian Grahammer, Christoph Wigge, Christoph Schell, et al.
Journal of Cachexia, Sarcopenia and Muscle
|
March 28, 2019
Compression of morbidity in a progeroid mouse model through the attenuation of myostatin/activin signalling
Khalid Alyodawi, Wilbert P Vermeij, Saleh Omairi, et al.
Plos Pathogens
|
April 21, 2022
Th17 cell plasticity towards a T-bet-dependent Th1 phenotype is required for bacterial control in Staphylococcus aureus infection
Patricia Bartsch, Christoph Kilian, Malte Hellmig, et al.
Acta Neuropathologica
|
January 20, 2024
Temporal change of DNA methylation subclasses between matched newly diagnosed and recurrent glioblastoma
Richard Drexler, Robin Khatri, Ulrich Schüller, et al.
Journal of Cachexia, Sarcopenia and Muscle
|
September 9, 2024
Improved health by combining dietary restriction and promoting muscle growth in DNA repair-deficient progeroid mice
Wilbert P Vermeij, Khalid Alyodawi, Ivar van Galen, et al.
The New England Journal of Medicine
|
May 14, 2020
Multiorgan and Renal Tropism of SARS-CoV-2
Victor G Puelles, Marc Lütgehetmann, Maja T Lindenmeyer, et al.
Kidney International
|
March 28, 2014
Renal fibrosis is the common feature of autosomal dominant tubulointerstitial kidney diseases caused by mutations in mucin 1 or uromodulin
Arif B Ekici, Thomas Hackenbeck, Vincent Morinière, et al.
Page
of 64