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Cell Death and Differentiation|September 17, 2002
The C-terminal activation domain of the STAT-1 transcription factor is necessary and sufficient for stress-induced apoptosisS Janjua, A Stephanou, D S LatchmanSeminars in Arthritis and Rheumatism|November 7, 1998
The role of in vitro expression systems in the investigation of antibodies to DNAA Rahman, D S Latchman, D A IsenbergSeminars in Arthritis and Rheumatism|January 1, 1999
Immunoglobulin variable region sequences of human monoclonal anti-DNA antibodiesA Rahman, D S Latchman, D A IsenbergAutoimmunity|January 1, 1994
Bcl-2 and Fas, molecules which influence apoptosis. A possible role in systemic lupus erythematosus?L M Rose, D S Latchman, D A IsenbergBritish Journal of Rheumatology|February 1, 1997
Apoptosis in peripheral lymphocytes in systemic lupus erythematosus: a reviewL M Rose, D S Latchman, D A IsenbergBritish Journal of Rheumatology|April 1, 1995
Bcl-2 expression is unaltered in unfractionated peripheral blood mononuclear cells in patients with systemic lupus erythematosusL M Rose, D S Latchman, D A IsenbergJournal of Molecular and Cellular Cardiology|April 5, 2003
SAG attenuates apoptotic cell death caused by simulated ischaemia/reoxygenation in rat cardiomyocytesA Chanalaris, Y Sun, D S Latchman, et al.The Journal of Rheumatology|February 1, 1994
The tissue specific elevation in synthesis of the 90 kDa heat shock protein precedes the onset of disease in lupus prone MRL/lpr miceG B Faulds, D A Isenberg, D S LatchmanLupus|January 1, 1997
Elevated soluble fas production in SLE correlates with HLA status not with disease activityL M Rose, D S Latchman, D A IsenbergJournal of Autoimmunity|April 1, 1989
Elevated levels of the 90 kd heat shock protein in a proportion of SLE patients with active diseaseP M Norton, D A Isenberg, D S LatchmanPageof 72