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B Lutterbach

Showing results (1-10 of 18) with videos related to

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Gene|March 16, 2000
Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiationB Lutterbach, S W Hiebert
Oncogene|February 27, 1997
Overexpression of c-Myc and cell immortalization alters c-Myc phosphorylationB Lutterbach, S R Hann
Journal of Cellular Biochemistry|February 18, 1999
c-Myc transactivation domain-associated kinases: questionable role for map kinases in c-Myc phosphorylationB Lutterbach, S R Hann
Molecular and Cellular Biology|August 1, 1994
Hierarchical phosphorylation at N-terminal transformation-sensitive sites in c-Myc protein is regulated by mitogens and in mitosisB Lutterbach, S R Hann
Current Opinion in Hematology|September 19, 2001
Role of co-repressors in transcriptional repression mediated by the t(8;21), t(16;21), t(12;21), and inv(16) fusion proteinsS W Hiebert, B Lutterbach, J Amann
Molecular and Cellular Biology|June 20, 1998
The MYND motif is required for repression of basal transcription from the multidrug resistance 1 promoter by the t(8;21) fusion proteinB Lutterbach, D Sun, J Schuetz, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 27, 1999
The inv(16) encodes an acute myeloid leukemia 1 transcriptional corepressorB Lutterbach, Y Hou, K L Durst, et al.
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|August 1, 1991
YACs and the C. elegans genomeA Coulson, Y Kozono, B Lutterbach, et al.
The Journal of Biological Chemistry|January 5, 2000
A mechanism of repression by acute myeloid leukemia-1, the target of multiple chromosomal translocations in acute leukemiaB Lutterbach, J J Westendorf, B Linggi, et al.
Oncogene|October 20, 2000
B-Myc is preferentially expressed in hormonally-controlled tissues and inhibits cellular proliferationM A Gregory, Q Xiao, G A Cornwall, et al.
Pageof 2

Showing results (1-10 of 18) with videos related to

Sort By:
Pageof 2
Gene|March 16, 2000
Role of the transcription factor AML-1 in acute leukemia and hematopoietic differentiationB Lutterbach, S W Hiebert
Oncogene|February 27, 1997
Overexpression of c-Myc and cell immortalization alters c-Myc phosphorylationB Lutterbach, S R Hann
Journal of Cellular Biochemistry|February 18, 1999
c-Myc transactivation domain-associated kinases: questionable role for map kinases in c-Myc phosphorylationB Lutterbach, S R Hann
Molecular and Cellular Biology|August 1, 1994
Hierarchical phosphorylation at N-terminal transformation-sensitive sites in c-Myc protein is regulated by mitogens and in mitosisB Lutterbach, S R Hann
Current Opinion in Hematology|September 19, 2001
Role of co-repressors in transcriptional repression mediated by the t(8;21), t(16;21), t(12;21), and inv(16) fusion proteinsS W Hiebert, B Lutterbach, J Amann
Molecular and Cellular Biology|June 20, 1998
The MYND motif is required for repression of basal transcription from the multidrug resistance 1 promoter by the t(8;21) fusion proteinB Lutterbach, D Sun, J Schuetz, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 27, 1999
The inv(16) encodes an acute myeloid leukemia 1 transcriptional corepressorB Lutterbach, Y Hou, K L Durst, et al.
Bioessays : News and Reviews in Molecular, Cellular and Developmental Biology|August 1, 1991
YACs and the C. elegans genomeA Coulson, Y Kozono, B Lutterbach, et al.
The Journal of Biological Chemistry|January 5, 2000
A mechanism of repression by acute myeloid leukemia-1, the target of multiple chromosomal translocations in acute leukemiaB Lutterbach, J J Westendorf, B Linggi, et al.
Oncogene|October 20, 2000
B-Myc is preferentially expressed in hormonally-controlled tissues and inhibits cellular proliferationM A Gregory, Q Xiao, G A Cornwall, et al.
Pageof 2