Showing results (1-10 of 11) with videos related to
Sort By:
Pageof 2
Nederlands Tijdschrift Voor Geneeskunde|June 14, 1997
[Retinoids: use in combating cancer]B M van der Leede, T M VroomMolecular Carcinogenesis|January 1, 1993
Retinoic acid receptor and retinoid X receptor expression in retinoic acid-resistant human tumor cell linesB M van der Leede, C E van den Brink, P T van der SaagThe Journal of Biological Chemistry|June 27, 1997
Novel progesterone target genes identified by an improved differential display technique suggest that progestin-induced growth inhibition of breast cancer cells coincides with enhancement of differentiationH A Kester, B M van der Leede, P T van der Saag, et al.Molecular and Cellular Endocrinology|May 16, 1997
CYP2C7 expression in rat liver and hepatocytes: regulation by retinoidsS Westin, E Sonneveld, B M van der Leede, et al.Molecular and Cellular Endocrinology|August 11, 1995
Differential regulation of AP1 activity by retinoic acid in hormone-dependent and -independent breast cancer cellsB van der Burg, R Slager-Davidov, B M van der Leede, et al.Molecular and Cellular Endocrinology|February 1, 1993
Retinoic acid resistance of estradiol-independent breast cancer cells coincides with diminished retinoic acid receptor functionB van der Burg, B M van der Leede, L Kwakkenbos-Isbrücker, et al.The American Journal of Pathology|June 1, 1996
Immunohistochemical analysis of retinoic acid receptor-alpha in human breast tumors: retinoic acid receptor-alpha expression correlates with proliferative activityB M van der Leede, J Geertzema, T M Vroom, et al.The Journal of Biological Chemistry|July 18, 1997
Autoinduction of retinoic acid metabolism to polar derivatives with decreased biological activity in retinoic acid-sensitive, but not in retinoic acid-resistant human breast cancer cellsB M van der Leede, C E van den Brink, W W Pijnappel, et al.Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|August 26, 1998
Human retinoic acid (RA) 4-hydroxylase (CYP26) is highly specific for all-trans-RA and can be induced through RA receptors in human breast and colon carcinoma cellsE Sonneveld, C E van den Brink, B M van der Leede, et al.Mutation Research. Genetic Toxicology and Environmental Mutagenesis|February 24, 2020
Analysis of historical negative control group data from the rat in vivo micronucleus assayD P Lovell, M Fellows, J Saul, et al.Pageof 2