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Current Opinion in Rheumatology
|
June 1, 1991
Disease-modifying antirheumatic drugs: gold, penicillamine, antimalarials, and sulfasalazine
B McConkey
British Medical Journal
|
September 6, 1980
Drugs and rheumatoid arthritis
B McConkey
British Journal of Rheumatology
|
April 1, 1990
It is said that elevated erythrocyte sedimentation rate and the elevation of C-reactive protein (CRP) may be indicators of continuing joint destruction in rheumatoid arthritis. What then is the explanation for joint destruction in some patients in whom there is no such apparent elevation of either the sedimentation rate of CRP?
B McConkey
British Medical Journal (Clinical Research Ed.)
|
November 14, 1981
Policeman's heel
B McConkey
British Medical Journal (Clinical Research Ed.)
|
June 12, 1982
Rheumatoid cervical myelopathy
B McConkey
Agents and Actions
|
September 1, 1976
New drugs for inflammation - a clinical viewpoint for their assessment
B McConkey
Lancet (London, England)
|
September 10, 1977
Alpha-1-antitrypsin and rheumatoid arthritis
L Walsh, B McConkey
The Quarterly Journal of Medicine
|
January 1, 1986
The effect of antirheumatic drugs on circulating immune complexes in rheumatoid arthritis
P J Forster, B McConkey
The Journal of Rheumatology
|
December 1, 1984
Rheumatoid arthritis: the effects of treatment with dapsone on hemoglobin
K A Grindulis, B McConkey
The Journal of Rheumatology
|
May 1, 1991
Rifampicin in rheumatoid arthritis
B McConkey, R D Situnayake
Page
of 7
Search research articles
Search
Showing results (11-20 of 66) with videos related to
Sort By:
Page
of 7
Current Opinion in Rheumatology
|
June 1, 1991
Disease-modifying antirheumatic drugs: gold, penicillamine, antimalarials, and sulfasalazine
B McConkey
British Medical Journal
|
September 6, 1980
Drugs and rheumatoid arthritis
B McConkey
British Journal of Rheumatology
|
April 1, 1990
It is said that elevated erythrocyte sedimentation rate and the elevation of C-reactive protein (CRP) may be indicators of continuing joint destruction in rheumatoid arthritis. What then is the explanation for joint destruction in some patients in whom there is no such apparent elevation of either the sedimentation rate of CRP?
B McConkey
British Medical Journal (Clinical Research Ed.)
|
November 14, 1981
Policeman's heel
B McConkey
British Medical Journal (Clinical Research Ed.)
|
June 12, 1982
Rheumatoid cervical myelopathy
B McConkey
Agents and Actions
|
September 1, 1976
New drugs for inflammation - a clinical viewpoint for their assessment
B McConkey
Lancet (London, England)
|
September 10, 1977
Alpha-1-antitrypsin and rheumatoid arthritis
L Walsh, B McConkey
The Quarterly Journal of Medicine
|
January 1, 1986
The effect of antirheumatic drugs on circulating immune complexes in rheumatoid arthritis
P J Forster, B McConkey
The Journal of Rheumatology
|
December 1, 1984
Rheumatoid arthritis: the effects of treatment with dapsone on hemoglobin
K A Grindulis, B McConkey
The Journal of Rheumatology
|
May 1, 1991
Rifampicin in rheumatoid arthritis
B McConkey, R D Situnayake
Page
of 7