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The Journal of Experimental Medicine|January 1, 1988
T cell dysfunction in the diabetes-prone BB rat. A role for thymic migrants that are not T cell precursorsH M Georgiou, A C Lagarde, D BellgrauDiabetes|January 1, 1989
Abdominal intratesticular islet-xenograft survival in ratsH P Selawry, K B Whittington, D BellgrauEuropean Journal of Immunology|July 1, 1992
Specificity of rat T cell receptor V beta chain usage in proliferative responses to staphylococcal enterotoxin BK S Sellins, D Bellgrau, D P GoldScandinavian Journal of Immunology|February 1, 1982
Natural killer cells do not belong to the recirculating lymphocyte populationM Zöller, D Bellgrau, I Axberg, et al.Diabetes|January 1, 1986
Islet allografts are destroyed by disease occurrence in the spontaneously diabetic BB ratS J Prowse, D Bellgrau, K J LaffertyHormone and Metabolic Research = Hormon- Und Stoffwechselforschung = Hormones Et Metabolisme|June 1, 1996
The diabetic BB rat. Neither Th1 nor Th2?D Bellgrau, D Stenger, C Richards, et al.Diabetes|January 1, 1994
Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB ratD Bellgrau, J M Redd, K S SellinsJournal of Virology|May 1, 1988
Recognition of adenovirus E1A gene products on immortalized cell surfaces by cytotoxic T lymphocytesD Bellgrau, T A Walker, J L CookDiabetologia|January 1, 1996
Resistance to tolerance induction in the diabetes-prone biobreeding rat as one manifestation of abnormal responses to superantigensK S Sellins, D P Gold, D BellgrauScience (New York, N.Y.)|September 18, 1981
Spontaneous diabetes in rats: destruction of islets is prevented by immunological toleranceA Naji, W K Silvers, D Bellgrau, et al.Pageof 6