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British Journal of Pharmacology|October 9, 2007
Direct suppression of autoreactive lymphocytes in the central nervous system via the CB2 receptorB N DittelArchivum Immunologiae Et Therapiae Experimentalis|January 5, 2001
Evidence that Fas and FasL contribute to the pathogenesis of experimental autoimmune encephalomyelitisB N DittelCurrent Molecular Medicine|March 20, 2002
Mechanisms of T cell receptor antagonism: implications in the treatment of diseaseB N DittelJournal of Immunology (Baltimore, Md. : 1950)|January 1, 1995
The growth response to IL-7 during normal human B cell ontogeny is restricted to B-lineage cells expressing CD34B N Dittel, T W LeBienJournal of Immunology (Baltimore, Md. : 1950)|November 22, 2000
Differential sensitivity to mutations in a single peptide by two TCRs having identical beta-chains and closely related alpha-chainsB N Dittel, C A JanewayImmunological Reviews|August 18, 1999
Transgenes and knockout mutations in animal models of type 1 diabetes and multiple sclerosisF S Wong, B N Dittel, C A JanewayJournal of Immunology (Baltimore, Md. : 1950)|June 3, 1999
Evidence for Fas-dependent and Fas-independent mechanisms in the pathogenesis of experimental autoimmune encephalomyelitisB N Dittel, R M Merchant, C A JanewayJournal of Autoimmunity|July 6, 2000
Relapsing and remitting experimental autoimmune encephalomyelitis in B cell deficient miceB N Dittel, T H Urbania, C A JanewayJournal of Immunology (Baltimore, Md. : 1950)|May 1, 1997
Peptide antagonists inhibit proliferation and the production of IL-4 and/or IFN-gamma in T helper 1, T helper 2, and T helper 0 clones bearing the same TCRB N Dittel, D B Sant'Angelo, C A JanewayPageof 3