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Blood|September 15, 1996
Vascular cell adhesion molecule-1 is involved in mediating hypoxia-induced sickle red blood cell adherence to endothelium: potential role in sickle cell diseaseB N Setty, M J StuartPediatric Pathology & Molecular Medicine|April 4, 2003
Hemostatic alterations in sickle cell disease: relationships to disease pathophysiologyM J Stuart, B N SettyCurrent Opinion in Hematology|February 27, 2001
Acute chest syndrome of sickle cell disease: new light on an old problemM J Stuart, B N SettyThe Journal of Clinical Investigation|January 1, 1986
15-Hydroxy-5,8,11,13-eicosatetraenoic acid inhibits human vascular cyclooxygenase. Potential role in diabetic vascular diseaseB N Setty, M J StuartBlood|September 9, 1999
Sickle cell acute chest syndrome: pathogenesis and rationale for treatmentM J Stuart, B N SettyThe Journal of Laboratory and Clinical Medicine|September 1, 1996
Sickle red blood cells stimulate endothelial cell production of eicosanoids and diacylglycerolB N Setty, D Chen, M J StuartBiochemical and Biophysical Research Communications|July 31, 1987
13-Hydroxyoctadecadienoic acid (13-HODE) stimulates prostacyclin production by endothelial cellsB N Setty, M Berger, M J StuartProstaglandins|November 1, 1987
The platelet cyclooxygenase metabolite 12-L-hydroxy-5, 8, 10-hepta-decatrienoic acid (HHT) may modulate primary hemostasis by stimulating prostacyclin productionP D Sadowitz, B N Setty, M StuartPediatric Research|July 1, 1995
Sickle cell vaso-occlusive crisis is associated with abnormalities in the ratio of vasoconstrictor to vasodilator prostanoidsB N Setty, D Chen, M J StuartPediatrics|May 1, 1985
Effect of changes in oxygen tension on vascular and platelet hydroxyacid metabolites. II. Hypoxia increases 15-hydroxyeicosatetraenoic acid, a proangiogenic metaboliteB N Setty, C Ganley, M J StuartPageof 3