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Journal of Immunotoxicology|October 30, 2008
Developmental exposure to the potent aryl hydrocarbon receptor agonist 2,3,7,8-tetrachlorodibenzo-p-dioxin Impairs the cell-mediated immune response to infection with influenza a virus, but enhances elements of innate immunityBeth A Vorderstrasse, Jennifer A Cundiff, B Paige LawrenceToxicological Sciences : an Official Journal of the Society of Toxicology|November 21, 2007
The aryl hydrocarbon receptor affects distinct tissue compartments during ontogeny of the immune systemJason P Hogaboam, Amanda J Moore, B Paige LawrenceReproductive Toxicology (Elmsford, N.Y.)|June 4, 2009
TCDD exposure disrupts mammary epithelial cell differentiation and functionLoretta L Collins, Betina J Lew, B Paige LawrenceCellular Immunology|January 30, 2004
Fewer CTL, not enhanced NK cells, are sufficient for viral clearance from the lungs of immunocompromised miceHaley D Neff-LaFord, Beth A Vorderstrasse, B Paige LawrenceClinical and Vaccine Immunology : CVI|July 13, 2012
Memory CD8+ T cells are sufficient to alleviate impaired host resistance to influenza A virus infection caused by neonatal oxygen supplementationMatthew Giannandrea, Min Yee, Michael A O'Reilly, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 13, 2012
Novel cellular targets of AhR underlie alterations in neutrophilic inflammation and inducible nitric oxide synthase expression during influenza virus infectionJennifer L Head Wheeler, Kyle C Martin, B Paige LawrenceChemico-Biological Interactions|July 26, 2005
Increased mortality associated with TCDD exposure in mice infected with influenza A virus is not due to severity of lung injury or alterations in Clara cell protein contentAndrea A Bohn, Kevin S Harrod, Sabine Teske, et al.American Journal of Respiratory Cell and Molecular Biology|December 22, 2012
Neonatal hyperoxia increases sensitivity of adult mice to bleomycin-induced lung fibrosisMin Yee, Bradley W Buczynski, B Paige Lawrence, et al.Journal of Immunology (Baltimore, Md. : 1950)|June 21, 2007
Aryl hydrocarbon receptor activation during influenza virus infection unveils a novel pathway of IFN-gamma production by phagocytic cellsHaley Neff-LaFord, Sabine Teske, Timothy P Bushnell, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|March 13, 2012
Lung development and the host response to influenza A virus are altered by different doses of neonatal oxygen in miceBradley W Buczynski, Min Yee, B Paige Lawrence, et al.Pageof 11