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Cellular Microbiology|May 1, 2014
The ability of an attaching and effacing pathogen to trigger localized actin assembly contributes to virulence by promoting mucosal attachmentEmily M Mallick, John J Garber, Vijay K Vanguri, et al.
BMC Gastroenterology|January 30, 2016
Novel exonic mutation inducing aberrant splicing in the IL10RA gene and resulting in infantile-onset inflammatory bowel disease: a case reportTadahiro Yanagi, Tatsuki Mizuochi, Yugo Takaki, et al.
Frontiers in Immunology|March 3, 2020
Alterations in T and B Cell Receptor Repertoires Patterns in Patients With IL10 Signaling Defects and History of Infantile-Onset IBDLael Werner, Yu Nee Lee, Erez Rechavi, et al.
Journal of Pediatric Gastroenterology and Nutrition|June 25, 2019
Risk-factors Associated With Poor Outcomes in VEO-IBD Secondary to XIAP Deficiency: A Case Report and Literature ReviewAsama Lekbua, Jodie Ouahed, Amy E O'Connell, et al.
The Journal of Pathology|April 20, 2018
Loss of N-WASP drives early progression in an Apc model of intestinal tumourigenesisHayley T Morris, Loic Fort, Heather J Spence, et al.
Blood|March 14, 2012
Wiskott-Aldrich syndrome protein (WASP) and N-WASP are critical for peripheral B-cell development and functionLisa S Westerberg, Carin Dahlberg, Marisa Baptista, et al.
Gastroenterology|September 20, 2011
Gut-tropic T cells that express integrin α4β7 and CCR9 are required for induction of oral immune tolerance in miceBarbara Cassani, Eduardo J Villablanca, Francisco J Quintana, et al.
Clinical Gastroenterology and Hepatology : the Official Clinical Practice Journal of the American Gastroenterological Association|March 22, 2021
A Systematic Review of Monogenic Inflammatory Bowel DiseaseRyusuke Nambu, Neil Warner, Daniel J Mulder, et al.
Blood|November 16, 2002
Gene therapy for Wiskott-Aldrich syndrome: rescue of T-cell signaling and amelioration of colitis upon transplantation of retrovirally transduced hematopoietic stem cells in miceChristoph Klein, Deanna Nguyen, Ching-Hui Liu, et al.
Immunity|June 11, 2004
Induced reactivity of intestinal CD4(+) T cells with an epithelial cell lectin, galectin-4, contributes to exacerbation of intestinal inflammationAkira Hokama, Emiko Mizoguchi, Ken Sugimoto, et al.
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