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B Viollet

Showing results (21-30 of 35) with videos related to

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Oncogene|April 20, 1999
Regulation of cdc2 gene expression by the upstream stimulatory factors (USFs)S North, X Espanel, F Bantignies, et al.
Bone|April 20, 2010
AMP-activated protein kinase (AMPK) activation regulates in vitro bone formation and bone massM Shah, B Kola, A Bataveljic, et al.
European Journal of Cancer (Oxford, England : 1990)|December 3, 2014
Lipoprotein internalisation induced by oncogenic AMPK activation is essential to maintain glioblastoma cell growthM Ríos, M Foretz, B Viollet, et al.
Diabetologia|September 28, 2011
Metformin activates AMP-activated protein kinase in primary human hepatocytes by decreasing cellular energy statusX Stephenne, M Foretz, N Taleux, et al.
Life Sciences|February 18, 2014
The AMPK-SIRT signaling network regulates glucose tolerance under calorie restriction conditionsM F P Silvestre, B Viollet, P W Caton, et al.
Osteoporosis International : a Journal Established As Result of Cooperation Between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA|May 7, 2013
The anti-diabetic drug metformin does not affect bone mass in vivo or fracture healingJ Jeyabalan, B Viollet, P Smitham, et al.
Acta Physiologica (Oxford, England)|June 18, 2014
Interleukin-6 deletion in mice driven by aP2-Cre-ERT2 prevents against high-fat diet-induced gain weight and adiposity in female miceB Navia, B Ferrer, M Giralt, et al.
Biochemical Society Transactions|January 28, 2003
A possible role for AMP-activated protein kinase in exercise-induced glucose utilization: insights from humans and transgenic animalsJ N Nielsen, S B Jørgensen, C Frøsig, et al.
Nature Medicine|January 1, 1996
Bcl-2 protects from lethal hepatic apoptosis induced by an anti-Fas antibody in miceV Lacronique, A Mignon, M Fabre, et al.
Molecular and Cellular Endocrinology|February 11, 2017
Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68]S C Chen, R Brooks, J Houskeeper, et al.
Pageof 4

Showing results (21-30 of 35) with videos related to

Sort By:
Pageof 4
Oncogene|April 20, 1999
Regulation of cdc2 gene expression by the upstream stimulatory factors (USFs)S North, X Espanel, F Bantignies, et al.
Bone|April 20, 2010
AMP-activated protein kinase (AMPK) activation regulates in vitro bone formation and bone massM Shah, B Kola, A Bataveljic, et al.
European Journal of Cancer (Oxford, England : 1990)|December 3, 2014
Lipoprotein internalisation induced by oncogenic AMPK activation is essential to maintain glioblastoma cell growthM Ríos, M Foretz, B Viollet, et al.
Diabetologia|September 28, 2011
Metformin activates AMP-activated protein kinase in primary human hepatocytes by decreasing cellular energy statusX Stephenne, M Foretz, N Taleux, et al.
Life Sciences|February 18, 2014
The AMPK-SIRT signaling network regulates glucose tolerance under calorie restriction conditionsM F P Silvestre, B Viollet, P W Caton, et al.
Osteoporosis International : a Journal Established As Result of Cooperation Between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA|May 7, 2013
The anti-diabetic drug metformin does not affect bone mass in vivo or fracture healingJ Jeyabalan, B Viollet, P Smitham, et al.
Acta Physiologica (Oxford, England)|June 18, 2014
Interleukin-6 deletion in mice driven by aP2-Cre-ERT2 prevents against high-fat diet-induced gain weight and adiposity in female miceB Navia, B Ferrer, M Giralt, et al.
Biochemical Society Transactions|January 28, 2003
A possible role for AMP-activated protein kinase in exercise-induced glucose utilization: insights from humans and transgenic animalsJ N Nielsen, S B Jørgensen, C Frøsig, et al.
Nature Medicine|January 1, 1996
Bcl-2 protects from lethal hepatic apoptosis induced by an anti-Fas antibody in miceV Lacronique, A Mignon, M Fabre, et al.
Molecular and Cellular Endocrinology|February 11, 2017
Corrigendum to "Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms" [Mol. Cell. Endocrinol. 440 15 January 2017 57-68]S C Chen, R Brooks, J Houskeeper, et al.
Pageof 4