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Biochemistry and Biophysics Reports|October 17, 2025
Hypoxia response in glioblastoma cells: effect of trehalose on macropinocytosis, autophagy and cell survivalBarbara Del Bello, Cristina Ulivieri, Emilia Maellaro
Cancer Cell International|July 21, 2022
The autophagy inducer trehalose stimulates macropinocytosis in NF1-deficient glioblastoma cellsBarbara Del Bello, Alessandra Gamberucci, Paola Marcolongo, et al.
Plos One|February 26, 2013
Cisplatin-induced apoptosis inhibits autophagy, which acts as a pro-survival mechanism in human melanoma cellsBarbara Del Bello, Marzia Toscano, Daniele Moretti, et al.
Archives of Biochemistry and Biophysics|October 12, 2014
Calpains and cancer: friends or enemies?Daniele Moretti, Barbara Del Bello, Giulia Allavena, et al.
Annals of the New York Academy of Sciences|March 23, 2004
Cisplatin-induced apoptosis in melanoma cells: role of caspase-3 and caspase-7 in Apaf-1 proteolytic cleavage and in execution of the degradative phasesBarbara Del Bello, Marta A Valentini, Mario Comporti, et al.
Biomacromolecules|March 4, 2009
Cisplatin/hydrogel complex in cancer therapyMario Casolaro, Renzo Cini, Barbara Del Bello, et al.
The Journal of Biological Chemistry|March 3, 2004
Loss of MYC confers resistance to doxorubicin-induced apoptosis by preventing the activation of multiple serine protease- and caspase-mediated pathwaysEmanuela Grassilli, Andrea Ballabeni, Emilia Maellaro, et al.
Colloids and Surfaces. B, Biointerfaces|August 6, 2011
Hydrogel containing L-valine residues as a platform for cisplatin chemotherapyMario Casolaro, Barbara Del Bello, Del Bello Barbara, et al.
Experimental Cell Research|January 20, 2004
Role of caspases-3 and -7 in Apaf-1 proteolytic cleavage and degradation events during cisplatin-induced apoptosis in melanoma cellsBarbara Del Bello, Marta A Valentini, Paola Mangiavacchi, et al.
Plos One|February 7, 2015
Calpain-3 impairs cell proliferation and stimulates oxidative stress-mediated cell death in melanoma cellsDaniele Moretti, Barbara Del Bello, Giulia Allavena, et al.
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